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Corticosteroid therapy-induced obesity in children

Clinical Pediatrics
|March 1, 1986
PubMed

Insights

Corticosteroid therapy for nephrotic syndrome can cause obesity in children. Persistent obesity is unlikely in normal-weight children who don't exceed 130% relative weight during treatment.

Area of Science:

  • Pediatric Endocrinology
  • Nephrology

Background:

  • Corticosteroids, like prednisone, are crucial for treating idiopathic nephrotic syndrome in children.
  • Obesity is a known side effect of corticosteroid therapy, but its persistence requires further investigation.

Purpose of the Study:

  • To evaluate the severity and persistence of corticosteroid-induced obesity in children treated for idiopathic nephrotic syndrome.
  • To identify risk factors for persistent obesity following long-term prednisone therapy.

Main Methods:

  • Retrospective analysis of 23 children (aged 1-14 years) treated with prednisone for over 60 days.
  • Tracking relative weight at therapy initiation, during therapy, and after cessation (at least 6 months post-therapy).

Main Results:

  • Peak relative weight increased during therapy (mean 119% vs. 107% at baseline).
  • Four children remained obese post-therapy; all had initial relative weight ≥110% and peak relative weight >130%.
  • Children with normal initial weight (<110%) and peak relative weight <130% did not experience persistent obesity.

Conclusions:

  • Persistent obesity after corticosteroid therapy for nephrotic syndrome is primarily a risk for children with higher initial or peak relative weights.
  • The risk of persistent obesity appears small in normal-weight children who maintain relative weights below 130% during treatment.

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