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Related Experiment Video

Updated: Jun 7, 2025

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage
10:34

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage

Published on: August 30, 2020

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High-Grade Subarachnoid Hemorrhage - Beyond Guidelines.

Sarah Wyckoff1, Sherry Hsiang-Yi Chou1

  • 1Department of Neurology, Northwestern University Feinberg School of Medicine, 625 North Michigan Avenue Suite 1150, Chicago IL 60611, USA.

Neurologic Clinics
|November 15, 2024
PubMed
Summary
This summary is machine-generated.

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High-grade subarachnoid hemorrhage (SAH) increases risks for acute respiratory distress syndrome (ARDS) and delayed cerebral ischemia (DCI). Current treatments for these SAH complications need further investigation for efficacy and safety.

Area of Science:

  • Neurosurgery
  • Critical Care Medicine
  • Neurology

Background:

  • Subarachnoid hemorrhage (SAH) presents with variable clinical severity.
  • High-grade SAH is associated with increased mortality and severe complications like ARDS and DCI.
  • Treating ARDS in SAH patients requires careful consideration of intracranial pressure and cerebral perfusion.

Purpose of the Study:

  • To review current treatment strategies for ARDS and DCI in SAH.
  • To highlight the challenges in managing these complications.
  • To emphasize the need for further research into effective therapies.

Main Methods:

  • Literature review of existing treatment approaches for ARDS and DCI in SAH.
  • Analysis of the impact of treatments on intracranial pressure and cerebral perfusion.
Keywords:
ARDSDelayed cerebral ischemiaIntracranial hypertensionPractice guidanceSubarachnoid hemorrhageVasospasm

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Last Updated: Jun 7, 2025

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage
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Published on: August 30, 2020

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Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model
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  • Evaluation of the efficacy and risk/benefit profiles of current interventions.

    • Main Results:

    • Treatment of ARDS in SAH necessitates specialized approaches due to ICP and CPP concerns.
    • DCI in SAH is a significant cause of morbidity, often not correlating with angiographic vasospasm.
    • Current therapeutic options for DCI and vasospasm require more research.

    Conclusions:

    • Nimodipine is the sole established therapeutic agent proven to improve SAH outcomes.
    • Further investigation is crucial to determine the efficacy and safety of treatments for ARDS and DCI in SAH.
    • Optimizing management of SAH complications remains a critical clinical challenge.