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Updated: Jun 6, 2025

A Murine Model of Stent Implantation in the Carotid Artery for the Study of Restenosis

A Murine Model of Stent Implantation in the Carotid Artery for the Study of Restenosis

Published on: May 14, 2013

In-stent restenosis.

Rym El Khoury¹, Ahmad Asha², Philip V Bystrom²

¹Division of Vascular Surgery, Department of Surgery, Endeavor Health/NorthShore University Health System, Evanston, IL, USA - relkhoury@northshore.org.

The Journal of Cardiovascular Surgery

|November 26, 2024

Summary

This summary is machine-generated.

Peripheral arterial occlusive disease treated with endovascular recanalization faces restenosis challenges, especially in femoropopliteal and tibial arteries. This review details the causes and treatments for restenosis after peripheral endovascular intervention.

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Last Updated: Jun 6, 2025

A Murine Model of Stent Implantation in the Carotid Artery for the Study of Restenosis

A Murine Model of Stent Implantation in the Carotid Artery for the Study of Restenosis

Published on: May 14, 2013

Imaging In-Stent Restenosis: An Inexpensive, Reliable, and Rapid Preclinical Model

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Published on: September 14, 2009

Implantation of Human-Sized Coronary Stents into Rat Abdominal Aorta Using a Trans-Femoral Access

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Published on: November 19, 2020

Area of Science:

Vascular Surgery
Interventional Cardiology
Endovascular Therapy

Background:

Endovascular arterial recanalization is a primary treatment for peripheral arterial occlusive disease.
While technically successful in over 90% of cases, long-term patency varies, particularly in femoropopliteal and tibial arteries.
Complex plaque, multiple lesions, and hemodynamic factors challenge sustained outcomes in peripheral endovascular interventions.

Conclusions:

Restenosis remains a significant limitation of peripheral endovascular intervention, impacting long-term outcomes.
Understanding the specific challenges of femoropopliteal and tibial arteries is crucial for improving treatment efficacy.
Further research and therapeutic innovations are needed to enhance the durability of endovascular treatments for peripheral arterial disease.