A stearate-rich diet and oleate restriction directly inhibit tumor growth via the unfolded protein response

Jumpei Ogura1, Koji Yamanoi2, Kentaro Ishida1

  • 1Department of Gynecology and Obstetrics, Kyoto University Graduate School of Medicine, Kyoto, Japan.

PubMed

Insights

Dietary stearic acid inhibits cancer growth by inducing DNA damage and apoptosis via the unfolded protein response (UPR) pathway. Modulating dietary fatty acids, increasing stearic acid and decreasing oleic acid, shows promise for cancer therapy.

Area of Science:

  • Oncology
  • Molecular Biology
  • Nutritional Science

Background:

  • Fatty acids influence cancer cell properties and are explored in cancer therapeutics.
  • Limited research exists on individual fatty acid effects and their interactions in cancer.

Purpose of the Study:

  • To investigate the impact of specific fatty acids, particularly stearic acid, on cancer cell growth and survival.
  • To explore the role of dietary interventions in modulating tumor growth.

Main Methods:

  • Analysis of individual fatty acid effects on cancer cells.
  • Utilizing a stearate-rich diet combined with stearoyl-CoA desaturase-1 inhibition in ovarian cancer models and patient-derived xenografts (PDXs).
  • Assessing tumor growth, DNA damage, apoptosis, and fatty acid levels (stearate and oleate) in tumors.

Main Results:

  • Stearic acid demonstrated a stronger inhibitory effect on cancer cell growth than palmitic acid, inducing DNA damage and apoptosis via the unfolded protein response (UPR).
  • The presence of oleic acid mitigated the negative effects of stearic acid.
  • Combined dietary intervention (increased stearate, decreased oleate) significantly reduced tumor growth in various models, including chemotherapy-resistant ones.

Conclusions:

  • Dietary stearic acid directly inhibits tumor growth through DNA damage and apoptosis mediated by the UPR pathway.
  • Dietary interventions manipulating stearic and oleic acid levels represent a potential therapeutic strategy for cancer treatment.
  • This research opens avenues for developing novel cancer therapies based on fatty acid modulation.

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