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Aortic Regurgitation I: Introduction

Aortic Regurgitation I: Introduction

IntroductionAortic regurgitation is characterized by the backward flow of blood from the aorta into the left ventricle during diastole and arises from the improper closure of the aortic valve. This condition results in left ventricular volume overload and can stem from both acute and chronic etiologies, each contributing uniquely to the disease's progression and symptomatology.Acute and Chronic CausesAcute aortic regurgitation often results from events that suddenly impair the integrity of the...

Aortic Regurgitation III: Medical Management

Aortic Regurgitation III: Medical Management

Aortic regurgitation (AR) is when the aortic valve does not close or seal properly, leading to backward blood circulation from the aorta into the left ventricle during diastole. Common causes of AR include rheumatic heart disease, congenital valve defects, and aortic root dilation. Managing AR requires a multifaceted approach to alleviate symptoms, preserve left ventricular function, and address the underlying cause of the regurgitation. Patients with symptomatic AR or significant left...

Aneurysm I: Introduction

Aneurysm I: Introduction

An aortic aneurysm is a localized outpouching or dilation at a weak point in the artery wall. It may involve different parts of the aorta, such as the abdominal aorta, aortic arch, or thoracic aorta.Etiological factorsSeveral disorders are associated with aortic aneurysms.Congenital causes, such as primary connective tissue disorders like Marfan syndrome, impact the integrity and strength of connective tissues, notably affecting the aorta. Marfan syndrome is a genetic disorder that specifically...

Aneurysm II: Clinical Manifestations and Diagnostic Studies

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Thoracic, aortic arch and abdominal aneurysms are significant vascular conditions that can present with various clinical manifestations and lead to serious complications. Understanding these manifestations and the appropriate diagnostic studies is essential for effective management and treatment.Thoracic Aortic AneurysmsThoracic aortic aneurysms often remain asymptomatic until they reach a size that impinges on adjacent structures. They typically cause deep, diffuse chest pain that radiates to...

Aneurysm III: Interprofessional Care

Aneurysm III: Interprofessional Care

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Aneurysm IV: Nursing Management

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Related Experiment Video

Updated: Jul 11, 2026

A New Murine Model of Endovascular Aortic Aneurysm Repair

A New Murine Model of Endovascular Aortic Aneurysm Repair

Published on: July 7, 2013

Type A Aortic Dissection After Thoracic Endovascular Aortic Repair for Type B.

Kyle A McCullough¹, Katherine R Hebeler², John B Eisenga³

¹Department of Cardiovascular Research, Baylor Scott & White Research Institute, Plano, Texas.

The American Journal of Cardiology

|December 11, 2024

Summary

Type B aortic dissection treated with thoracic endovascular aortic repair (TEVAR) can be followed by a new Type A aortic dissection (TAAD). Late TAAD after TEVAR for Type B dissection appears to be a distinct event.

Keywords:

TEVAR aortic dissection

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Novel and Innovative Hybrid Technique for Type A Aortic Dissection

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Novel and Innovative Hybrid Technique for Type A Aortic Dissection

Novel and Innovative Hybrid Technique for Type A Aortic Dissection

Published on: March 28, 2025

Area of Science:

Cardiovascular Surgery
Vascular Surgery
Thoracic Surgery

Background:

Type A aortic dissection (TAAD) is a serious cardiovascular condition.
Thoracic endovascular aortic repair (TEVAR) is used for aortic dissections and aneurysms.
The association between prior TEVAR for Type B dissection and subsequent TAAD is not well understood.

Purpose of the Study:

To investigate patients who underwent TEVAR for Type B aortic dissection followed by TAAD.
To analyze the characteristics and outcomes of TAAD occurring after TEVAR for Type B dissection.

Main Methods:

Retrospective review of 171 consecutive patients undergoing open ascending aortic repair for TAAD over 6 years.
Identification of patients with a history of TEVAR for Type B aortic dissection.
Analysis of the interval between TEVAR and TAAD, and the location of the primary tear.

Main Results:

Nine (5.2%) of 171 TAAD patients had prior TEVAR for Type B dissection.
The mean interval from TEVAR to TAAD repair was 4.6 years.
In most cases, the TAAD entry tear was remote from the previous endograft, suggesting a new event.

Conclusions:

Type A aortic dissection occurring late after TEVAR for Type B dissection is uncommon.
These late TAAD events appear to be spontaneous, new occurrences rather than complications of the prior TEVAR.
Further research is needed to understand the specific mechanisms and risk factors for this sequence of aortic events.