KDM5C and KDM5D mutations have different consequences in clear cell renal cell carcinoma cells

Marvin Müller1, Kyra Zodel1, Behnaz A Abhari1

  • 1Department of Internal Medicine I, Hematology, Oncology and Stem Cell Transplantation, Faculty of Medicine, Medical Center-University of Freiburg, Freiburg, Germany.

Communications Biology
|February 15, 2025
PubMed

Insights

KDM5C mutations are common in male clear cell renal cell carcinomas (ccRCC). Loss of the Y chromosome, including KDM5D, also occurs in male ccRCC, impacting tumor development and gene expression differently than expected.

Area of Science:

  • Oncology
  • Genetics
  • Molecular Biology

Background:

  • KDM5C mutations are prevalent in male clear cell renal cell carcinoma (ccRCC) but rare in females.
  • The Y chromosome, containing the KDM5C homolog KDM5D, is often lost in male ccRCC with KDM5C mutations.

Purpose of the Study:

  • To investigate the context-dependent functions of KDM5C in ccRCC.
  • To explore the interplay between KDM5C mutation and Y chromosome loss in male ccRCC development.

Main Methods:

  • Introducing KDM5C mutations into ccRCC cell lines.
  • Analyzing xenograft tumor formation and transcriptional consequences.
  • Investigating KDM5C and KDM5D genomic binding sites and gene expression regulation.

Main Results:

  • KDM5C mutations induced distinct phenotypes and transcriptional changes in male and female ccRCC cell lines.
  • Loss of Y chromosome (and KDM5D) in male cells prevented tumor formation, a phenotype rescued by KDM5C co-mutation.
  • KDM5C and KDM5D regulate overlapping and distinct gene sets, with functions not solely explained by direct promoter/enhancer activity.

Conclusions:

  • KDM5C and KDM5D exhibit both shared and unique functions, challenging the notion of equivalent roles to a second KDM5C allele in females.
  • KDM5C mutation and Y chromosome loss interact in male ccRCC development, highlighting sex-specific mechanisms in tumorigenesis.

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