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Related Concept Videos

Glaucoma: Overview01:25

Glaucoma: Overview

Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
Open Angle Glaucoma: Treatment01:27

Open Angle Glaucoma: Treatment

In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
Drugs such as carbonic anhydrase inhibitors, α2- and...
Angle Closure Glaucoma: Treatment01:28

Angle Closure Glaucoma: Treatment

Angle-closure glaucoma, or closed-angle glaucoma, is an eye condition where the iris bulges out and blocks the iridocorneal angle, resulting in a buildup of aqueous humor and increased intraocular pressure. Immediate medical attention is necessary due to the sudden onset of symptoms. The treatment for angle-closure glaucoma includes short-term and long-term approaches. Short-term treatment involves using eye drops like pilocarpine to lower intraocular pressure by increasing aqueous humor...

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Baseline Optical Coherence Tomography Biomarkers Associated with the 2-Year Development of Macular Atrophy or Fibrosis in Neovascular Age-related Macular Degeneration.

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Switching to aflibercept 8 mg in neovascular age-related macular degeneration: real-world outcomes according to switch indication.

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Comment to: Hashiya N, Maruko I, Maruko R et al. Increased intraocular pressure immediately after aflibercept 8 mg intravitreal injection for neovascular age-related macular degeneration. Graefes Arch Clin Exp Ophthalmol (2026) 264:1611-1617. doi:10.1007/s00417-026-07117-3.

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1 MONTH VS. 3 MONTHS HEAVY SILICONE OIL TAMPONADE IN COMPLEX RHEGMATOGENOUS RETINAL REDETACHMENT.

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Rotating the Intraocular Lens to Prevent Posterior Capsular Opacification in Cataract Surgeries
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DELAYED RHEGMATOGENOUS RETINAL DETACHMENT AFTER MACULAR SURGERY: A Multicentric Study.

Sumit Randhir Singh1, Francesco Boscia2, Giacomo Boscia2

  • 1Akhand Jyoti Eye Hospital, CoE Mastichak, Saran, Bihar, India.

Retina (Philadelphia, Pa.)
|February 25, 2025
PubMed
Summary

Delayed rhegmatogenous retinal detachment (RRD) after macular surgery, often due to missed retinal breaks, occurred in 0.55% of cases. Despite successful reattachment, visual acuity frequently worsened post-RRD repair.

Keywords:
epiretinal membranefull-thickness macular holeoptical coherence tomographyretinal breakrhegmatogenous retinal detachment

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Area of Science:

  • Ophthalmology
  • Retinal Surgery
  • Macular Surgery

Background:

  • Macular surgery, including procedures for full-thickness macular holes (FTMH) and epiretinal membranes (ERM), can be complicated by delayed rhegmatogenous retinal detachment (RRD).
  • Understanding the incidence, timing, and characteristics of RRD following macular surgery is crucial for patient management and surgical planning.

Purpose of the Study:

  • To investigate the anatomical and visual outcomes of eyes that develop delayed RRD after undergoing macular surgery.
  • To identify the causes and patterns of retinal breaks associated with RRD after macular procedures.

Main Methods:

  • A retrospective, multicentric study analyzed eyes with RRD repair occurring at least 3 months post-macular surgery (FTMH or ERM).
  • Data collected included pre- and postoperative best-corrected visual acuity (BCVA), surgical details, time to RRD, tamponade, and retinal break characteristics.

Main Results:

  • RRD developed in 16 of 2,906 eyes (0.55%) after a mean of 8.4 months post-macular surgery.
  • Missed or de novo retinal breaks were the predominant cause (68.75%), followed by probable microbreaks, giant retinal tears, and retinoschisis-related breaks.
  • Anatomical success (reattachment after one surgery) was achieved in 87.5% of eyes, but BCVA significantly worsened from 20/80 to 20/160 (P < 0.001).

Conclusions:

  • Delayed RRD after macular surgery is uncommon but significant, often resulting from missed or new retinal breaks.
  • While anatomical reattachment rates are high, a substantial proportion of patients experience a decline in visual acuity despite successful surgery.