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Published on: July 29, 2012
Dichlorvos poisoning caused chicken cerebellar autophagy and changes of Caecal microflora
Juan Chen1, Yun Zhang2, Chenxi Jiang1
1Jiangxi Provincial Key Laboratory for Animal Health, Institute of Animal Population Health, College of Animal Science and Technology, Jiangxi Agricultural University, Nanchang 330045, PR China.
Abstract:
In order to explore the effects of acute dichlorvos exposure on cerebellar autophagy and cecal microbes in broilers and to analyze the relationship between autophagy-related genes and cecal microbes. Broilers were randomly divided into three groups (with 16 broilers in each group)and respectively given distilled water and dichlorvos (2.48 mg / kg, 11.3 mg / kg). The cecal contents and cerebellum samples were collected after poisoning symptoms of broilers, and the antioxidant indexes such as SOD and CAT in cerebellum were detected. Hematoxylin-eosin (HE) staining of cerebellum and cecum, and immunofluorescence sections of cerebellum LC3 were made. RT-PCR and western blot were used to detect the expression of oxidative stress and autophagy-related genes in cerebellar tissue. The cecal contents were analyzed by 16S rRNA high-throughput sequencing, and then the correlation between the expression of autophagy-related genes and the abundance of intestinal microbes was analyzed. It was concluded that dichlorvos exposure destroyed the normal morphological structure of the cerebellum and cecum in broilers, which induced oxidative stress and autophagy in the cerebellum of broilers, reduced the diversity of cecal microorganisms, and destroyed the steady state of the cecal microbial structure. In addition, The changes of mRNA expression of autophagy-related genes is related to some specific bacteria. In summary, this study found that dichlorone exposure can cause cerebellar oxidative stress and autophagy, and the mechanism of cerebellar injury in broilers is linked to cecal microbiota changes, potentially offering a new direction for researching dichlorone's pathogenic mechanism.
Insights
Acute dichlorvos exposure harms broiler cerebellum and cecum structure, inducing oxidative stress and autophagy. This pesticide exposure also disrupts cecal microbes, linking gene expression changes to specific bacteria and suggesting a new pathogenic mechanism for dichlorvos injury.
Area of Science:
- Veterinary Toxicology
- Microbiology
- Cellular Biology
Background:
- Dichlorvos is an organophosphate pesticide with known toxicity.
- Autophagy plays a role in cellular stress responses.
- The gut microbiome's influence on host health is increasingly recognized.
Purpose of the Study:
- To investigate the impact of acute dichlorvos exposure on broiler cerebellar autophagy and cecal microbiota.
- To analyze the relationship between autophagy-related gene expression and cecal microbial composition.
Main Methods:
- Broilers were exposed to varying doses of dichlorvos.
- Cerebellar and cecal tissues were analyzed using histological staining and immunofluorescence.
- Gene expression of oxidative stress and autophagy markers was quantified via RT-PCR and Western blot.
- Cecal microbial communities were profiled using 16S rRNA high-throughput sequencing.
Main Results:
- Dichlorvos exposure caused structural damage to the cerebellum and cecum.
- Oxidative stress and autophagy were induced in the broiler cerebellum.
- Cecal microbial diversity and structure were significantly reduced.
- Changes in autophagy-related gene expression correlated with specific bacterial populations.
Conclusions:
- Acute dichlorvos exposure induces cerebellar oxidative stress and autophagy in broilers.
- Dichlorvos-induced cerebellar injury is associated with alterations in the cecal microbiota.
- The interplay between dichlorvos, cerebellar autophagy, and gut microbiota offers a novel perspective on its toxicological mechanisms.
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