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Trace elements and alcohol.

W W Dinsmore, D McMaster, M E Callender

    The Science of the Total Environment
    |March 15, 1985
    PubMed
    Summary
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    Alcohol consumption is linked to lower zinc levels and higher copper, iron, and ferritin in patients with and without cirrhosis. Alcohol may enhance zinc absorption in healthy individuals.

    Area of Science:

    • Biochemistry
    • Nutritional Science
    • Hepatology

    Background:

    • Alcohol abuse is a significant global health issue.
    • Cirrhosis is a severe consequence of chronic alcohol consumption.
    • Trace element imbalances are common in alcoholic liver disease.

    Purpose of the Study:

    • To investigate serum zinc, copper, iron, and ferritin levels in alcoholic patients with and without cirrhosis.
    • To assess selenium status in these patient groups.
    • To determine the effect of alcohol on zinc absorption in healthy volunteers.

    Main Methods:

    • Serum samples were collected from alcoholic patients (with and without cirrhosis) and healthy volunteers.
    • Levels of zinc, copper, iron, ferritin, and selenium were measured.

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  • Zinc absorption was assessed in healthy volunteers after single-dose alcohol and zinc administration.

    • Main Results:

    • Alcoholic patients, with or without cirrhosis, exhibited decreased serum zinc.
    • Elevated serum copper, iron, and ferritin were observed in alcoholic patients.
    • No selenium deficiency was detected in either alcoholic group.
    • Co-administration of alcohol and zinc in normal volunteers increased serum zinc, suggesting enhanced absorption.

    Conclusions:

    • Alcohol consumption significantly alters trace element metabolism, particularly zinc, copper, and iron, in patients with and without cirrhosis.
    • Alcohol may play a role in increasing zinc absorption.
    • Selenium levels appear unaffected by alcohol consumption in the studied groups.