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Risk factors of ventricular fibrillation during rapid amphotericin B infusion

Insights

Rapid amphotericin B infusions can cause dangerous hyperkalemia and arrhythmias in patients with kidney problems. Slowing infusion rates or using hemodialysis prevents these severe adverse events.

Area of Science:

  • Pharmacology
  • Nephrology
  • Cardiology

Background:

  • Amphotericin B is known to cause potassium loss in vitro and arrhythmias in dogs.
  • Human cases of amphotericin B-induced hyperkalemic ventricular arrhythmias are rare.
  • This study investigates the safety of amphotericin B infusion in patients with impaired renal function.

Observation:

  • A patient with anuria experienced ventricular fibrillation and severe hyperkalemia during rapid, high-dose amphotericin B infusion.
  • Prolonged infusion (3 hours) and concurrent hemodialysis prevented hyperkalemia and arrhythmias in anuric patients.
  • Lower peak serum concentrations of amphotericin B were observed with slower infusions or during dialysis.

Findings:

  • Rapid amphotericin B infusion (1.4 mg/kg over 45 min) led to peak serum concentrations of 6.7 µg/ml and ventricular fibrillation in an anuric patient.
  • Slower infusions (3-4 hours) or hemodialysis during infusion maintained normal serum potassium levels and prevented arrhythmias.
  • Patients with normal renal function receiving lower doses over 45 min showed only minor potassium increases.

Implications:

  • Rapid amphotericin B infusion is associated with significant risk of hyperkalemia and ventricular arrhythmias in patients with impaired potassium excretion.
  • Hemodialysis and careful potassium monitoring are crucial for anuric patients receiving amphotericin B.
  • Adjusting infusion rates and considering concurrent hemodialysis can mitigate the risk of severe adverse cardiac events.

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