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Hypercalcemia in leprosy.

E Ryzen, F R Singer

Archives of Internal Medicine

|July 1, 1985

Summary

This summary is machine-generated.

This study details a rare case of hypercalcemia in leprosy, where vitamin D and parathyroid hormone levels were low. Corticosteroid treatment effectively resolved the high calcium levels without malignancy.

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Area of Science:

Endocrinology
Infectious Diseases
Dermatology

Background:

Leprosy, a chronic infectious disease, can present with diverse clinical manifestations.
Hypercalcemia is an uncommon complication of leprosy, necessitating investigation into its underlying mechanisms.

Observation:

A patient with leprosy presented with symptomatic hypercalcemia.
Laboratory findings revealed suppressed aminoterminal parathyroid hormone and 25-hydroxy-cholecalciferol levels.
Elevated urinary hydroxyproline concentrations were noted, suggesting increased bone resorption.

Findings:

The patient's hypercalcemia was not attributed to malignancy.
The biochemical profile indicated a non-classical cause for elevated calcium.
Treatment with corticosteroids led to the resolution of hypercalcemia.

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Implications:

This case highlights the importance of considering endocrine and metabolic derangements in leprosy patients.
Corticosteroids may be an effective therapeutic option for managing hypercalcemia in specific leprosy-related contexts.
Further research is warranted to elucidate the pathophysiology of hypercalcemia in leprosy.