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Headache in transient ischemic attacks (TIA).

C Loeb, C Gandolfo, D Dall'Agata

    Cephalalgia : an International Journal of Headache
    |May 1, 1985
    PubMed
    Summary
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    Headache is a common symptom in transient ischemic attacks (TIA), particularly in vertebrobasilar cases. Headache characteristics can help differentiate TIA subtypes and predict arterial occlusion.

    Area of Science:

    • Neurology
    • Neuroscience
    • Vascular Neurology

    Background:

    • Transient ischemic attacks (TIA) present diagnostic challenges, especially regarding headache evaluation.
    • The classical TIA definition requires supplementation with normal CT scans to prevent misdiagnosis.

    Purpose of the Study:

    • To analyze the clinical features of headache in patients with transient ischemic attacks (TIA).
    • To investigate the relationship between headache characteristics and TIA location (carotid vs. vertebrobasilar).
    • To explore the association of headache with arterial occlusion and CT findings in TIA patients.

    Main Methods:

    • Retrospective analysis of clinical data from 90 patients with TIA who underwent contrast-enhanced CT scans.
    • Detailed recording of headache occurrence, location, timing, and relationship to neurological symptoms.

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  • Correlation of headache patterns with TIA laterality, vascular territory, presence of arterial occlusion, and CT scan results.

    • Main Results:

    • Headache was present in 30% of TIA patients, more frequent in vertebrobasilar TIAs than carotid TIAs.
    • Headache often preceded or accompanied other neurological symptoms (77.8%).
    • Ipsilateral headache was anterior in carotid TIAs and posterior in vertebrobasilar TIAs. Headache preceded neurological symptoms in arterial occlusion cases.

    Conclusions:

    • Headache is a significant clinical feature in TIA, offering clues to the underlying pathology.
    • Headache characteristics, including location and timing, can aid in differentiating TIA subtypes and identifying arterial occlusion.
    • Further research into the pathophysiology of TIA-associated headache is warranted.