Deletion of PPARα in mouse brown adipocytes increases their De Novo Lipogenesis

  • 1Université Côte d'Azur, CNRS, Inserm, Adipocible Research Study Group, Institut de Biologie Valrose (iBV), Nice, France.
  • 2Institute of Metabolic and Cardiovascular Diseases, I2MC, University of Toulouse, Inserm, Toulouse III University - Paul Sabatier (UPS), Toulouse, France; Department of Medical Biochemistry, Toulouse University Hospitals.
  • 3Université Côte d'Azur, CNRS, Inserm, IRCAN, Nice, France.
  • 4Institute of Metabolic and Cardiovascular Diseases, I2MC, University of Toulouse, Inserm, Toulouse III University - Paul Sabatier (UPS), Toulouse, France.
  • 5Université Côte d'Azur, CNRS, Laboratoire de PhysioMédecine Moléculaire (LP2M), Laboratories of Excellence Ion Channel Science and Therapeutics, Nice, France.
  • 6Université Côte d'Azur, CNRS, Inserm, Adipocible Research Study Group, Institut de Biologie Valrose (iBV), Nice, France; Rheumatology Department, Hospital Pasteur 2 CHU, Adipocible Research Study Group, 06000 Nice, France.
  • 7Toxalim (Research Centre in Food Toxicology), Université de Toulouse, INRAE, ENVT, INP-Purpan, UPS, Toulouse, France.
  • 8Université Paris Cité, Institut Cochin, CNRS, Inserm, Paris, France.
  • 9Laboratory of Translational Nutrition Biology, Institute of Food, Nutrition and Health, ETH Zurich, Schwerzenbach, Switzerland.
  • 10Institute of Metabolic and Cardiovascular Diseases, I2MC, University of Toulouse, Inserm, Toulouse III University - Paul Sabatier (UPS), Toulouse, France; Department of Medical Biochemistry, Toulouse University Hospitals; Institut Universitaire de France (IUF), Paris, France.
  • 11Université Côte d'Azur, CNRS, Inserm, Adipocible Research Study Group, Institut de Biologie Valrose (iBV), Nice, France. Electronic address: Isabelle.SATNEY@univ-cotedazur.fr.

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Abstract

OBJECTIVES

Peroxisome Proliferator-Activated Receptors (PPARs) are nuclear receptors involved in the control of lipid metabolism. The PPARα isoform is highly expressed in brown adipose tissue (BAT). However, its precise role in BAT remains unclear. Here, we aimed to investigate the role of PPARα in BAT of high fat diet-induced obese mice in a thermoneutral environment.

METHODS

We used tamoxifen-inducible-BAT specific PPARα knockout mice (PPARαBATKO) that were housed at thermoneutrality to minimize BAT basal activation, fed a high-fat diet for 20 weeks and challenged with a β3-adrenergic agonist (CL316,243) during the last week. Both male and female mice were studied.

RESULTS

Body weight and glucose tolerance tests were similar in both sexes and genotypes. However, BAT morphology was altered in PPARαBATKO mice, with more unilocular and larger lipid droplets compared to control mice, suggesting BAT impaired function. Indeed, when treated with CL316,243, both male and female mice had increased de novo lipogenesis (DNL), reflected by an increased expression of ChREBPβ and lipogenic enzymes ACLY, ACC1, FASN and SCD1. These changes were accompanied by an increase in fatty acids in triglycerides, and thus an increase in lipid storage. Moreover, lipid profiles in phospholipids were different, suggesting a modification in the membrane content with an increase of palmitoleate.

CONCLUSIONS

Altogether, our results reveal a key role for PPARα in DNL in BAT and in the regulation of lipid metabolism in HFD-induced obesity.

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