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Updated: Sep 13, 2025

Development and Maintenance of a Preclinical Patient Derived Tumor Xenograft Model for the Investigation of Novel Anti-Cancer Therapies
Published on: September 30, 2016
Integrated transcriptomic and functional modeling reveals AKT and mTOR synergy in colorectal cancer
Marcin Duleba1, Eliza Zimoląg2, Joanna Szuszkiewicz2
1Ryvu Therapeutics, Kraków, Poland. marcin.duleba@ryvu.com.
Abstract:
Colorectal cancer (CRC) treatment remains challenging due to genetic heterogeneity and resistance mechanisms. To address this, we developed a drug discovery pipeline using patient-derived primary CRC cultures with diverse genomic profiles. These cultures closely resemble certain molecular characteristics of primary and metastatic CRC, highlighting their promise as a translational platform for therapeutic evaluation. Importantly, our engineered model and patient-derived cells reflect the complexity and heterogeneity of primary tumors, not observed with standard immortalized cell lines, offering a more clinically relevant system, although further validation is needed. High-throughput screening (HTS) of 4255 compounds identified 33 with selective efficacy against CRC cells, sparing normal, healthy epithelial cells. Among the tested combinations, everolimus (mTOR inhibitor) and uprosertib (AKT inhibitor) demonstrated promising synergy at clinically relevant concentrations, with favorable therapeutic windows confirmed across tested patient-derived cultures. Notably, this synergy, revealed through advanced models, might have been overlooked in traditional immortalized cell lines, highlighting the translational advantage of patient-derived systems. Furthermore, the integration of machine learning into the HTS pipeline significantly improved scalability, cost-efficiency, and predictive accuracy. Our findings underscore the potential of patient-derived materials combined with machine learning-enhanced drug discovery to advance personalized therapies. Specifically, mTOR-AKT inhibition emerges as a promising strategy for CRC treatment, paving the way for more effective and targeted therapeutic approaches.
Insights
Developing patient-derived colorectal cancer models and using machine learning identified novel drug combinations. mTOR-AKT inhibition shows promise for personalized colorectal cancer therapy.
Area of Science:
- Oncology
- Drug Discovery
- Genomics
Background:
- Colorectal cancer (CRC) treatment is hindered by genetic diversity and drug resistance.
- Standard cell lines may not fully represent primary tumor complexity and heterogeneity.
Purpose of the Study:
- To develop a translational drug discovery platform using patient-derived CRC cultures.
- To identify novel therapeutic strategies for colorectal cancer through high-throughput screening and machine learning.
Main Methods:
- Established patient-derived primary colorectal cancer cultures reflecting tumor heterogeneity.
- Conducted high-throughput screening (HTS) of 4255 compounds.
- Integrated machine learning into the HTS pipeline for enhanced efficiency and accuracy.
- Evaluated drug combinations, including everolimus (mTOR inhibitor) and uprosertib (AKT inhibitor).
Main Results:
- Identified 33 compounds with selective efficacy against colorectal cancer cells.
- Discovered synergistic effects between everolimus and uprosertib at clinically relevant concentrations.
- Demonstrated the translational advantage of patient-derived models over traditional cell lines.
- Machine learning integration improved HTS scalability, cost-effectiveness, and predictive power.
Conclusions:
- Patient-derived models combined with machine learning accelerate personalized colorectal cancer therapy development.
- mTOR-AKT inhibition represents a promising targeted strategy for colorectal cancer treatment.
- Advanced models are crucial for uncovering synergistic drug interactions potentially missed by conventional methods.
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