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Lactic acidosis and beer drinking.

J R Evans, J Main, P E Mitchell

    Scottish Medical Journal
    |October 1, 1985
    PubMed
    Summary
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    Excessive beer consumption can lead to Wernicke-Korsakoff syndrome, lactic acidosis, and hyponatremia. Prompt thiamine treatment rapidly corrected acidosis and hyponatremia, though some Wernicke-Korsakoff symptoms persisted.

    Area of Science:

    • Neurology
    • Toxicology
    • Nutritional Medicine

    Background:

    • Chronic excessive alcohol consumption, particularly beer and lager, is a known risk factor for nutritional deficiencies.
    • Thiamine (Vitamin B1) deficiency is strongly associated with neurological disorders like Wernicke-Korsakoff syndrome.

    Observation:

    • A case study detailing a patient with severe lactic acidosis, hyponatremia, and Wernicke-Korsakoff syndrome symptoms.
    • The patient's condition was linked to long-term, excessive intake of beer and lager.

    Findings:

    • Intramuscular thiamine administration resulted in rapid resolution of lactic acidosis.
    • Intravenous therapy effectively corrected the patient's hyponatremia.
    • Despite treatment, the patient exhibited persistent chronic neurological deficits characteristic of Wernicke-Korsakoff syndrome upon discharge.

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    Implications:

    • Highlights the critical role of thiamine in preventing and managing alcohol-related neurological and metabolic complications.
    • Underscores the potential for severe, irreversible neurological damage even with timely metabolic correction.
    • Emphasizes the need for awareness regarding the risks associated with excessive consumption of specific alcoholic beverages like beer and lager.