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Neurotoxicity, α-synuclein pathology, and mitochondrial dysfunction: A comparative study of different mouse models of

Xiwen Tang1, Yifei He1, Min Liang2

  • 1Department of Translational Neuroscience, Jing'an District Center Hospital of Shanghai, State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, Institutes of Brain Science, Fudan University, Shanghai, China.

Neural Regeneration Research
|September 29, 2025
PubMed
Abstract

No abstract available in PubMed .

Keywords:
1-methyl-4-phenyl-1236-tetrahydropyridine-induced modelMitoPark modelferroptosis pathwayglial cell activationinterleukin 17 pathwaymitochondrial dysfunctionnerve regenerationneurodegenerationnigrostriatal pathwaypS129-α-synα-syn A53T transgenic model

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