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Hypoglycemia and Glucagon

Hypoglycemia and Glucagon

Without prolonged fasting, healthy individuals maintain blood glucose levels above 3.5 mM due to a well-adapted neuroendocrine counterregulatory system that effectively prevents acute hypoglycemia, a potentially life-threatening condition. The primary clinical scenarios for hypoglycemia encompass diabetes treatment, inappropriate production of endogenous insulin or insulin-like substances by tumors, and the use of glucose-lowering agents in non-diabetic individuals. Notably, hypoglycemia in the...

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Related Experiment Video

Updated: Jan 16, 2026

Measuring Lactase Enzymatic Activity in the Teaching Lab

Measuring Lactase Enzymatic Activity in the Teaching Lab

Published on: August 6, 2018

Methylprednisolone-Induced Hyperlactatemia: A Case Report.

Sarah Saenz¹, Anila Bhatti¹, Aaron Beck²

¹Department of Anesthesiology, University of Wisconsin Hospital and Clinics, Madison, WI, USA.

|September 29, 2025

Summary

This summary is machine-generated.

Methylprednisolone can cause lactic acidosis in critically ill patients. This case highlights a rare, non-ischemic type B lactic acidosis, emphasizing the need for differential diagnosis in intensive care units.

Area of Science:

Critical Care Medicine
Neurology
Pharmacology

Background:

Lactic acidosis is a common critical care complication, often indicating severe conditions like end-organ ischemia.

Keywords:

Lactic acidosis Resuscitation Spinal cord injury Steroids

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While typically associated with hypoperfusion (Type A), other causes of lactic acidosis exist, necessitating thorough investigation.