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Mode of host cell penetration by bacteriophage phi X174

Journal of Virology
|June 1, 1971
PubMed

Insights

Bacteriophage phiX174 attaches to bacterial cell walls, releasing its DNA. Specific treatments reveal attachment sites and prevent DNA release, offering insights into phage-host interactions.

Area of Science:

  • Microbiology
  • Virology
  • Molecular Biology

Background:

  • Bacteriophage phiX174 is an icosahedral phage.
  • It lacks a complex tail assembly for host cell attachment.

Purpose of the Study:

  • To investigate the attachment mechanism of bacteriophage phiX174 to bacterial host cells.
  • To understand the role of cell wall components and phage structure in DNA release.

Main Methods:

  • Incubation of phiX174 virions with isolated bacterial cell walls.
  • Treatment of cell walls with chloroform, lysozyme, and virions with formaldehyde.
  • Freeze-etch electron microscopy of adsorbed phages on Escherichia coli.
  • Radioactive labeling of phage proteins and recovery from spheroplasts.

Main Results:

  • Virions attached to cell wall fragments, leading to phage deoxyribonucleic acid (DNA) release.
  • Chloroform treatment prevented attachment; lysozyme or formaldehyde treatment prevented DNA release but not attachment.
  • Lysozyme treatment released structures that bound phiX174 at vertices, but without DNA release.
  • Phages adsorbed to bacteria were submerged in the cell wall with their fivefold axis normal to the surface.
  • Phage proteins were recovered as low-molecular-weight material and in progeny viruses.

Conclusions:

  • Bacteriophage phiX174 attachment involves specific interactions with bacterial cell wall components.
  • DNA release is a distinct step following attachment, dependent on cell wall integrity and phage viability.
  • The phage's fivefold axis of symmetry plays a role in its orientation and interaction with the host cell surface.

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