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ACPA prevents lung fibroblast-to-CAF transformation by reprogramming the tumor microenvironment through NSCLC-derived

Özge Boyacıoğlu1, Berfin Deniz Kalali1,2, Tuba Reçber3

  • 1Department of Medical Biochemistry, Faculty of Medicine, Atılım University, 06830, Ankara, Turkey.

Scientific Reports
|November 29, 2025
PubMed
Abstract

No abstract available in PubMed .

Keywords:
Arachidonylcyclopropylamide (ACPA)Cancer-associated fibroblasts (CAFs)ExosomesNon-small cell lung cancerTumor microenvironment (TME)

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