Related Experiment Video
Updated: May 6, 2026

Microscopic Replantation of Penile Glans Amputation Due to Circumcision
Published on: June 3, 2022
Necrotizing Cellulitis of the Finger Caused by Ring Embedment Leading to Amputation: A Case Report and Literature
Shreya Guha1, Manya Bali1, Zohaer Muttalib1
1Medical School, California Northstate University College of Medicine, Elk Grove, USA.
Abstract:
Constrictive ring embedment can cause progressive ischemia, tissue necrosis, and infection. Although embedded rings have been reported, digital amputation as a direct result is rarely documented. The authors present the case of a 62-year-old woman who presented to the emergency department with a five-day history of right ring finger cellulitis secondary to prolonged ring erosion extending to the bone. She reported pain, fever, and weakness, and was tachycardic with signs of sepsis. A physical exam revealed crepitus, though radiographs showed no subcutaneous air. Given the severity of ischemic tissue damage and the risk of reperfusion injury, the patient was transferred to the orthopedic service for digital amputation. The Braden scale, a measurement tool for pressure ulcer risk, identified her as high-risk for pressure ulcers, emphasizing the need for early intervention. Prolonged ring pressure had led to capillary occlusion, ischemia, and necrosis, creating an environment for bacterial infection. Ischemia-driven hypoxia further impaired bacterial clearance and wound healing, while reperfusion injury complicated management. This case highlights the importance of early recognition and intervention in ring-related ischemic injuries. Clinicians should be aware of the potential for severe vascular compromise, infection, and reperfusion injury in constrictive ring embedment.
Related Concept Videos
Necrosis
Morphological Manifestations of Necrosis
Necrotic cells show different types of morphological appearance depending on the type of tissue and infection. In coagulative necrosis, cells become...
Endocarditis II: Clinical Features of Infective Endocarditis
Cellular Injury IV: Necrosis
Diabetic Foot Ulcer