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Updated: Mar 31, 2026

Development of a Rabbit Chronic-Like Rotator Cuff Injury Model for Study of Fibrosis and Muscular Fatty Degeneration

Development of a Rabbit Chronic-Like Rotator Cuff Injury Model for Study of Fibrosis and Muscular Fatty Degeneration

Published on: March 31, 2023

Retroperitoneal fibrosis.

Augusto Vaglio¹, Francesco Peyronel², Ingeborg M Bajema³

¹Department of Biomedical, Experimental and Clinical Sciences "Mario Serio", University of Florence, Florence, Italy; Nephrology and Dialysis Unit, Meyer Children's Hospital IRCCS, Florence, Italy.

Lancet (London, England)

|March 29, 2026

Summary

This summary is machine-generated.

Retroperitoneal fibrosis is a rare immune condition causing tissue buildup around the aorta. Treatment involves medication like glucocorticoids and B-cell therapies, but relapses are common.

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Area of Science:

Immunology
Nephrology
Rheumatology

Background:

Retroperitoneal fibrosis (RPF) is a rare immune-mediated condition characterized by fibro-inflammatory tissue encasing retroperitoneal structures.
It can be idiopathic, associated with IgG4-related disease, or secondary to other conditions like infections or malignancies.
RPF pathogenesis involves complex immune cell interactions and can lead to severe complications affecting the kidneys and vasculature.

Purpose of the Study:

To provide a contemporary overview of retroperitoneal fibrosis.
To focus on the pathophysiology, differential diagnosis, and management of the disease.

Main Methods:

Review of current literature on retroperitoneal fibrosis.
Synthesis of information regarding etiology, immunopathogenesis, clinical presentation, and treatment strategies.

Main Results:

Idiopathic RPF has multifactorial origins, including genetic and environmental factors.
The immunopathogenesis involves intricate crosstalk between lymphocytes, macrophages, and other immune cells.
Complications include kidney injury and vascular occlusion, necessitating medical and sometimes surgical interventions.

Conclusions:

Medical therapy, including glucocorticoids and B-cell-targeting agents, is the mainstay of RPF treatment.
Despite effective treatments, relapses are frequent after treatment cessation.
A comprehensive understanding of RPF's pathophysiology and differential diagnosis is crucial for optimal management.