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Gender differences in the association between adverse childhood experiences and impulsivity in Saudi Arabian adults:
1Department of Psychiatry, College of Medicine, Imam Mohammad Ibn Saud Islamic University (IMSIU), Riyadh, Saudi Arabia.
Background:
Adverse childhood experiences (ACEs) are linked to impulsivity, a transdiagnostic risk factor for psychopathology. However, gender differences in ACE-impulsivity pathways remain unclear, particularly in Middle Eastern populations, and prior research has often conflated differential exposure with differential vulnerability while lacking rigorous multiple testing corrections.
Objective:
This study examined gender differences in ACE exposure, multidimensional impulsivity, and ACE-impulsivity associations in Saudi Arabian adults, distinguishing between differential exposure and differential vulnerability hypotheses using rigorous statistical methods.
Methods:
A nationwide cross-sectional online survey recruited 379 Saudi adults (222 female, 157 male; majority aged 18-25 years [55.7%]). Participants completed the Arabic 10-item ACE questionnaire and Short UPPS-P Impulsive Behavior Scale (five dimensions: Negative Urgency, Positive Urgency, Sensation Seeking, Lack of Premeditation, Lack of Perseverance). Analyses employed Benjamini-Hochberg false discovery rate (FDR) correction (q = .05) across test families, bootstrap confidence intervals (5,000 iterations) for all effect sizes, and hierarchical regression models testing Gender × ACE interactions with demographic covariates.
Results:
Females reported significantly higher ACE exposure (M = 2.24 vs. 1.75, p = .005), concentrated in emotional neglect (35.1% vs. 17.8%, FDR-significant) and household mental illness (29.3% vs. 16.6%, FDR-significant). No impulsivity dimension showed significant gender differences after FDR correction. Gender-stratified analyses revealed significant ACE correlations for four dimensions in females (ρ = 0.142-0.262) but only Negative Urgency in males (ρ = 0.222). However, bootstrap tests of cross-gender correlation differences were all non-significant (all Δρ 95% CIs included zero). Covariate-adjusted Gender × ACE interactions were uniformly null (all p > .16, ΔR² < 0.005).
Conclusions:
Gender differences in ACE-impulsivity associations reflect differential exposure (higher female ACE burden) rather than differential vulnerability (different per-ACE impact). Findings support universal trauma-informed interventions with ACE-type-specific modifications rather than fundamentally gender-differentiated approaches. Results highlight the importance of rigorous statistical controls and distinction between exposure and vulnerability in ACE research.
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