Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Cerebral Edema ll: Pathophysiology01:22

Cerebral Edema ll: Pathophysiology

Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this barrier loses...
Brain Abscess l: Introduction01:26

Brain Abscess l: Introduction

A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...
Cerebral Edema l: Introduction01:19

Cerebral Edema l: Introduction

Cerebral edema is a pathological increase in brain water content that disrupts intracranial pressure regulation and impairs neurological function. Because the cranial vault is rigid, even modest increases in tissue volume can compromise cerebral perfusion, distort neural structures, and initiate secondary injury. Cerebral edema develops through four principal mechanisms: vasogenic, cytotoxic, interstitial, and ionic.Vasogenic EdemaVasogenic edema arises from disruption of the blood–brain...
Encephalitis l: Introduction01:19

Encephalitis l: Introduction

Encephalitis is inflammation of the brain parenchyma, most often due to infections or autoimmune processes. It presents with neuropsychiatric features such as fever, altered mental status, behavioral changes, cognitive dysfunction, seizures, focal deficits, and sometimes autonomic instability. In some cases, the meninges are also involved, resulting in meningoencephalitis.Infectious CausesInfectious encephalitis is most commonly viral but can also result from bacterial, fungal, or parasitic...
Encephalitis ll: Pathophysiology01:26

Encephalitis ll: Pathophysiology

Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...
Hepatic Encephalopathy01:29

Hepatic Encephalopathy

DefinitionHepatic encephalopathy is a reversible neurologic syndrome that results from advanced liver dysfunction or portosystemic shunting. It leads to disturbances in cognition, behavior, and motor function due to the brain’s exposure to gut-derived toxins that the liver fails to detoxify.EtiologyThis condition develops either in the setting of acute fulminant hepatitis or progressively during chronic liver disease, such as cirrhosis and portal hypertension. Portosystemic shunting—including...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Dehydroepiandrosterone Sulfate/Cortisol Ratio as a Biomarker of Depressive Symptoms in Late-Onset Hypogonadism: An Analysis of 3,005 Males.

The world journal of men's health·2026
Same author

Evaluating the Precision of Intra-Spline Bipolar Potentials With Pentaspline Pulsed Field Ablation Catheter.

Journal of arrhythmia·2026
Same author

Impact of Diabetes on the Efficacy of Low-Voltage Area Ablation for Persistent Atrial Fibrillation: A Subanalysis of the SUPPRESS-AF Trial.

Journal of the American Heart Association·2026
Same author

Efficacy of Low-Voltage Area Ablation Across Substrate Size in Persistent Atrial Fibrillation: A Post Hoc Analysis of the SUPPRESS-AF Randomized Trial.

Journal of the American Heart Association·2026
Same author

[Spinal Epidural Abscess].

Brain and nerve = Shinkei kenkyu no shinpo·2026
Same author

Complete Low-Voltage Area Ablation in Persistent Atrial Fibrillation - Post Hoc Subanalysis of the SUPPRESS-AF Trial.

Circulation journal : official journal of the Japanese Circulation Society·2026

Related Experiment Video

Updated: May 21, 2026

Transient Transduction of the Strobilated Forms of Echinococcus granulosus
13:25

Transient Transduction of the Strobilated Forms of Echinococcus granulosus

Published on: September 16, 2022

[Cerebral Echinococcosis].

Tomohiro Uemura1, Takashi Kanda

  • 1Department of Neurology, Sibetsu City Hospital.

Brain and Nerve = Shinkei Kenkyu No Shinpo
|May 19, 2026
PubMed
Summary
This summary is machine-generated.

Cerebral echinococcosis, a rare but serious brain infection from Echinococcus tapeworms, poses diagnostic and treatment challenges. Early suspicion and surgical intervention are key for better outcomes in this CNS parasitic disease.

More Related Videos

Isolation And Dendritic Cell-Uptake of Small Extracellular Vesicles from Echinococcus granulosus
09:04

Isolation And Dendritic Cell-Uptake of Small Extracellular Vesicles from Echinococcus granulosus

Published on: March 28, 2025

Induction and Micro-CT Imaging of Cerebral Cavernous Malformations in Mouse Model
05:12

Induction and Micro-CT Imaging of Cerebral Cavernous Malformations in Mouse Model

Published on: September 4, 2017

Related Experiment Videos

Last Updated: May 21, 2026

Transient Transduction of the Strobilated Forms of Echinococcus granulosus
13:25

Transient Transduction of the Strobilated Forms of Echinococcus granulosus

Published on: September 16, 2022

Isolation And Dendritic Cell-Uptake of Small Extracellular Vesicles from Echinococcus granulosus
09:04

Isolation And Dendritic Cell-Uptake of Small Extracellular Vesicles from Echinococcus granulosus

Published on: March 28, 2025

Induction and Micro-CT Imaging of Cerebral Cavernous Malformations in Mouse Model
05:12

Induction and Micro-CT Imaging of Cerebral Cavernous Malformations in Mouse Model

Published on: September 4, 2017

Area of Science:

  • Neurology
  • Infectious Diseases
  • Parasitology

Background:

  • Cerebral echinococcosis is a rare, life-threatening central nervous system infection caused by Echinococcus tapeworms.
  • It typically results from metastasis from other infected organs, rarely presenting as a primary brain lesion.
  • Distinguishing cerebral echinococcosis from brain tumors is diagnostically challenging.

Purpose of the Study:

  • To highlight the diagnostic difficulties and treatment limitations of cerebral echinococcosis.
  • To emphasize the importance of clinical suspicion for accurate diagnosis.
  • To review current therapeutic strategies for this parasitic CNS infection.

Main Methods:

  • Literature review of cerebral echinococcosis cases.
  • Analysis of diagnostic challenges and treatment outcomes.
  • Focus on differentiation from malignant brain tumors.

Main Results:

  • Diagnosis is often delayed due to non-specific symptoms and rarity.
  • Surgical resection is the primary treatment modality.
  • Chemotherapy with benzimidazole antiparasitics shows limited efficacy.

Conclusions:

  • Cerebral echinococcosis requires high clinical suspicion for timely diagnosis.
  • Multidisciplinary approaches are necessary for effective management.
  • Further research into improved therapeutic agents is warranted.