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Updated: Jun 30, 2026

Quantification of three DNA Lesions by Mass Spectrometry and Assessment of Their Levels in Tissues of Mice Exposed to Ambient Fine Particulate Matter
Published on: May 29, 2019
DNA Structure-Dependent Enrichment of Oxidative Lesions
Maha Zewail-Foote1, Alex W Klattenhoff2, Karen M Vasquez2
1Department of Chemistry and Biochemistry, Southwestern University, 1001 E University Ave, Georgetown, Texas 78626, United States.
None:
Oxidative DNA lesions such as 8-oxo-7,8-dihydroguanine (8-oxoG) and abasic (AP) sites contribute to mutagenesis and genetic instability. H-DNA, a naturally occurring intramolecular triplex structure, promotes genetic instability and is susceptible to oxidative damage. Here, we examined how oxidative lesions distribute within an H-DNA-forming sequence. Although oxidative stress increased oxidized purines overall, no template-dependent differences were detected at a distal B-DNA locus. In contrast, AP sites and FPG-sensitive lesions were enriched within the H-DNA region relative to the B-DNA control, even under basal conditions. These findings indicate that H-DNA topology enhances local susceptibility to oxidative damage and shapes the spatial distribution of oxidative DNA lesions that can contribute to mutagenesis.
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