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Recombination between guanidine-resistant and dextran sulfate-resistant mutants of type 1 poliovirus

Journal of Virology
|March 1, 1969
PubMed

Insights

Recombination between poliovirus mutants generated more guanidine-resistant, dextran sulfate-resistant particles than expected by chance. This suggests genetic exchange, but dextran sulfate resistance wasn't linked to HBB resistance.

Area of Science:

  • Virology
  • Molecular Biology
  • Genetics

Background:

  • Poliovirus LSc2ab strain mutants were used to study genetic recombination.
  • One mutant was resistant to guanidine (gua(r)), the other to dextran sulfate and HBB (dex(r)HBB(r)).

Purpose of the Study:

  • To investigate genetic recombination in poliovirus.
  • To determine if resistance markers (dextran sulfate and HBB) are linked during recombination.

Main Methods:

  • Mixed infection of monkey kidney cells with two distinct poliovirus mutants.
  • Quantification of progeny virus particles with combined resistance markers (gua(r)dex(r)).
  • Analysis of HBB resistance in guanidine-resistant, dextran sulfate-resistant (gua(r)dex(r)) clones.

Main Results:

  • Progeny virus showed a 7-10 fold excess of gua(r)dex(r) particles compared to spontaneous mutation rates.
  • Recombination is proposed as the mechanism explaining the high yield of doubly mutant particles.
  • Analysis of 50 gua(r)dex(r) clones indicated that dextran sulfate resistance is not associated with HBB resistance during recombination.

Conclusions:

  • Genetic recombination occurs in poliovirus LSc2ab strain.
  • Dextran sulfate resistance and HBB resistance are independently assorted genetic markers in poliovirus recombination.

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