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Polyoma pseudovirions. II. Influence of host cell on pseudovirus production

Journal of Virology
|November 1, 1973
PubMed

Insights

Host cell type and infection duration impact polyoma virus production. Primary mouse embryo cells favor pseudovirion production, linked to DNA fragmentation, unlike other cell types.

Area of Science:

  • Virology
  • Molecular Biology
  • Cell Biology

Background:

  • Polyoma virus infection involves the production of both infectious virions and non-infectious pseudovirions.
  • The host cell environment and cellular processes play a critical role in viral replication and particle assembly.

Purpose of the Study:

  • To investigate how host cell type and infection duration influence the production ratio of polyoma pseudovirions to infectious polyoma virions.
  • To explore the correlation between host cell DNA fragmentation and pseudovirion production.

Main Methods:

  • Infection of different host cell types (primary mouse embryo cells, baby mouse kidney cells, 3T3D cells) with polyoma virus.
  • Analysis of the relative quantities of pseudovirions and polyoma virions produced over varying infection times.
  • Assessment of host cell DNA fragmentation patterns and sizes.

Main Results:

  • Primary mouse embryo cells predominantly produced pseudovirions, while baby mouse kidney and 3T3D cells yielded mainly infectious polyoma virus.
  • Extended infection times led to a higher proportion of pseudovirions relative to polyoma virions.
  • Pseudovirion production correlated with host cell DNA fragmentation to approximately 3 x 10(6) daltons, with more extensive fragmentation observed in primary mouse embryo cells.

Conclusions:

  • Host cell type is a key determinant in the ratio of pseudovirion to polyoma virion production.
  • Prolonged viral infection promotes pseudovirion formation, associated with significant host DNA fragmentation.
  • The extent of host DNA fragmentation is a critical factor influencing pseudovirion yield in polyoma virus infections.

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