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Autophagy induction in atrophic muscle cells requires ULK1 activation by TRIM32 through unanchored K63-linked polyubiquitin chains.

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AMBRA1 is able to induce mitophagy via LC3 binding, regardless of PARKIN and p62/SQSTM1.

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[Clinical indications for the use of cardiac MRI. By the SIRM Study Group on Cardiac Imaging].

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Qingfei Dayuan granules alleviate the inflammatory response in lipopolysaccharide-induced acute lung injury mice by inhibiting the Nf-κB signaling pathway and regulating the complement pathway.

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Atorvastatin potentiates the efficacy of gemcitabine in diabetic pancreatic cancer via remodeling the tumor immune microenvironment and attenuating vascular endothelial.

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Frontiers in cell and developmental biology·2026

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Biochemical Society transactions·2026

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Nature communications·2026

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Autophagy·2026

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Cardiac catecholamine levels and blood pressure after chronic treatment with beta-adrenergic blocking agents

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Intermediate dose enoxaparin in hospitalized patients with moderate-severe COVID-19: a pilot phase II single-arm study, INHIXACOVID19.

Predictive comorbidities of hospital admission in 1,571 SARS- CoV-2 positive patients: analysis of administrative data from an Italian Local Health Autority.

Autophagy induction in atrophic muscle cells requires ULK1 activation by TRIM32 through unanchored K63-linked polyubiquitin chains.

Modification of translation factor aIF5A from Sulfolobus solfataricus.

AMBRA1 is able to induce mitophagy via LC3 binding, regardless of PARKIN and p62/SQSTM1.

[Clinical indications for the use of cardiac MRI. By the SIRM Study Group on Cardiac Imaging].

Chrysin preserves renal structure and mitochondrial quality control in D-galactose-induced kidney aging: enhanced efficacy via nano-encapsulation.

Qingfei Dayuan granules alleviate the inflammatory response in lipopolysaccharide-induced acute lung injury mice by inhibiting the Nf-κB signaling pathway and regulating the complement pathway.

Phytochemical-mediated targeting of signaling pathways in glioblastoma: preclinical evidence, translational potential, and emerging nano-based therapeutic approaches.

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Cardiac catecholamine levels and blood pressure after chronic treatment with beta-adrenergic blocking agents

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