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[Pathogenesis of symptomatic facial hemiatrophy]
Zhurnal Nevropatologii I Psikhiatrii Imeni S.S. Korsakova (Moscow, Russia : 1952)
|January 1, 1979
Summary
Facial hemiatrophy can result from damage to the brainstem and superior cervical sympathetic ganglion. These neurological conditions affect both vegetative and somatic systems, leading to facial asymmetry.
Area of Science:
- Neurology
- Neuroanatomy
- Pathophysiology
Context:
- Presents clinical and electroencephalographic data from 19 patients.
- Examines cases of facial hemiatrophy following specific neurological insults.
Purpose:
- To investigate the pathogenetic relationship between neurological lesions and symptomatic facial hemiatrophy.
- To identify the role of central and peripheral nervous system structures in facial hemiatrophy development.
Summary:
- Facial hemiatrophy developed in patients with conditions including superior cervical sympathetic ganglionitis, stem encephalitis, Gasserian ganglion tumors, and syringobulbia.
- Lesions in the cerebral (mesencephalotruncal) and extracerebral (superior cervical sympathetic ganglion) structures were implicated.
- A connection between damage to vegetative and somatic nervous system components and facial hemiatrophy is established.
Impact:
- Highlights the complex interplay between central and peripheral nervous system damage in facial hemiatrophy.
- Provides insights into the neuroanatomical basis of facial hemiatrophy.
- Informs potential diagnostic and therapeutic strategies for facial hemiatrophy.