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Sensitivity of gentamicin-resistant Enterobacteriaceae to cefamandole and cefoxitin

Chemotherapy
|January 1, 1979
PubMed

Insights

Gentamicin-resistant bacteria often show increased resistance to cefamandole, with resistance genes transferable between species. This linked resistance is common, even where cefamandole isn't used.

Area of Science:

  • Microbiology
  • Antimicrobial Resistance
  • Molecular Biology

Background:

  • Enterobacteriaceae are common causes of healthcare-associated infections.
  • Antibiotic resistance, particularly to gentamicin, is a growing concern.
  • The relationship between gentamicin and cefamandole resistance in Enterobacteriaceae is not fully understood.

Purpose of the Study:

  • To investigate the co-occurrence and transferability of gentamicin and cefamandole resistance in Enterobacteriaceae.
  • To compare cefamandole and cefoxitin resistance patterns in gentamicin-resistant versus gentamicin-sensitive strains.

Main Methods:

  • Phenotypic characterization of antibiotic resistance in clinical isolates.
  • Conjugation experiments to assess the transferability of resistance genes.
  • Species identification and characterization of Enterobacteriaceae.

Main Results:

  • Gentamicin-resistant Enterobacteriaceae exhibited significantly higher resistance to cefamandole compared to gentamicin-sensitive strains.
  • Simultaneous transfer of cefamandole and gentamicin resistance via conjugation was demonstrated from multiple Enterobacteriaceae species.
  • Linked resistance plasmids for cefamandole and gentamicin were prevalent in the hospital environment, despite cefamandole's absence.
  • No significant difference in cefoxitin resistance was observed between gentamicin-resistant and sensitive isolates, and cefoxitin resistance was not transferable via conjugation.

Conclusions:

  • Plasmids carrying linked cefamandole and gentamicin resistance are common in Enterobacteriaceae within healthcare settings.
  • The presence of these plasmids suggests a potential for co-selection and spread of resistance, even without direct antibiotic pressure.
  • Cefoxitin resistance appears to be mediated by different mechanisms and is not directly linked to gentamicin resistance transfer.

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