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The morbidity of carotid endarterectomy
Insights
Carotid endarterectomy offers good long-term results for stroke prevention. However, objective assessment is crucial as asymptomatic arterial occlusion can occur post-surgery.
Area of Science:
- Vascular Surgery
- Neurology
- Clinical Outcomes
Background:
- Carotid endarterectomy is a procedure to remove plaque from carotid arteries.
- Stroke and transient ischemic attacks (TIAs) are often caused by carotid artery disease.
Purpose of the Study:
- To analyze the outcomes of 72 carotid endarterectomy operations.
- To evaluate the long-term efficacy and complications of the procedure.
Main Methods:
- Retrospective analysis of 72 carotid endarterectomy cases.
- Follow-up assessment over 1-7 years, including clinical evaluation and postoperative angiography.
Main Results:
- 76% of patients achieved satisfactory long-term results, remaining stroke-free.
- 13.8% experienced early postoperative neurological deficits; 5.5% transient and 8.3% permanent.
- 4.2% of reconstructions were found to be occluded on follow-up angiography, with some patients remaining asymptomatic.
Conclusions:
- Carotid endarterectomy can be effective in preventing recurrent stroke and TIAs.
- Postoperative asymptomatic arterial occlusion necessitates objective assessment methods.
- Increased surgical experience and routine use of intra-arterial shunting and angiography may reduce complication rates.
Abstract:
The results of 72 carotid endarterectomy operations carried out by three consultant surgeons were analysed. Thirty-eight operations (32.8 per cent) were performed in patients with mild or resolving stroke and 33 operations (45.9 per cent) in patients with transient ischaemic attacks and/or amaurosis fugax. Five patients (7 per cent) died in the early postoperative period, 2 as a result of strokes and the others from cardiac causes. Seventy-six per cent of patients had a satisfactory long term result, remaining free from storke, blindness or recurrent transient ischaemic attacks over the follow-up period of 1--7 years. There were 10 patients (13.8 per cent), including 2 who died, who developed early postoperative neurological deficits; 4 (5.5 per cent) had transient defects and 6 (8.3 per cent) had permanent deficits. Of these patients, 4 had proved occlusions of the reconstructed artery. In 3 patients, cerebral embolization during operation was suspected as the cause. There were, in addition, 2 asymptomatic patients whose reconstruction was shown to be occluded on follow-up postoperative angiography. The continued absence of symptoms, therefore, is no guarantee that the reconstruction is patent and some more objective method of assessment is necessary if the results of carotid endarterectomy are to be critically analysed. The incidence of neurological deficits could, it is believed, be reduced by greater experience with the operation, the routine use of peroperative intra-arterial shunting and peroperative angiography to confirm the patency of the reconstruction.
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