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Mice lacking inducible nitric oxide synthase have improved left ventricular contractile function and reduced apoptotic cell death late after myocardial infarction.

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Left ventricular diastolic dysfunction during demand ischemia: rigor underlies increased stiffness without calcium-mediated tension. Amelioration by glycolytic substrate.

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Related Experiment Videos

Limitations of lactate production as an index of myocardial ischemia

Related Concept Videos

Digitalis for treatment of congestive heart failure in patients in sinus rhythm.

Digitalis for treatment of congestive heart failure in patients in sinus rhythm.

Infusion of light chains from patients with cardiac amyloidosis causes diastolic dysfunction in isolated mouse hearts.

Mice lacking inducible nitric oxide synthase have improved left ventricular contractile function and reduced apoptotic cell death late after myocardial infarction.

Exercise training attenuates age-associated diastolic dysfunction in rats.

Left ventricular diastolic dysfunction during demand ischemia: rigor underlies increased stiffness without calcium-mediated tension. Amelioration by glycolytic substrate.

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