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Plaque formation by mumps virus and inhibition by antiserum

Applied Microbiology
|February 1, 1970
PubMed

Insights

Mumps virus strains Boston, ABC, and Ricki form larger plaques than the Jeryl Lynn vaccine strain. DEAE-dextran enhances plaque visualization, and plaque reduction assays effectively detect mumps virus neutralizing antibodies.

Area of Science:

  • Virology
  • Cell Biology

Background:

  • Mumps virus characterization is crucial for vaccine development and diagnostics.
  • Plaque assays are a standard method for quantifying viral infectivity.

Purpose of the Study:

  • To compare plaque formation characteristics of different mumps virus strains.
  • To evaluate the effect of DEAE-dextran on plaque visualization.
  • To assess the correlation between plaque assays and infectivity titrations.

Main Methods:

  • Culturing mumps virus strains (Boston, ABC, Ricki, Jeryl Lynn) in BGM cell monolayers.
  • Analyzing plaque size, morphology, and visualization with and without DEAE-dextran.
  • Performing simultaneous titrations using plaque formation and roller tube infectivity.
  • Demonstrating virus neutralization by antibody through plaque reduction assays.

Main Results:

  • Boston, ABC, and Ricki strains produced 1.0 mm plaques; Jeryl Lynn produced 0.3 mm plaques.
  • Ricki strain exhibited unique red plaques.
  • DEAE-dextran (400 µg/ml) improved visualization of Jeryl Lynn and Ricki plaques.
  • Plaque formation and roller tube titrations showed equal sensitivity.
  • Plaque reduction assays confirmed virus neutralization and antibody titer increases.

Conclusions:

  • Distinct plaque characteristics differentiate mumps virus strains.
  • DEAE-dextran aids in visualizing plaques from specific strains.
  • Plaque assays are reliable for mumps virus quantification and neutralization studies.

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