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Neuropathy in tetanus.

M Shahani, F D Dastur, D H Dastoor

    Journal of the Neurological Sciences
    |October 1, 1979
    PubMed
    Summary
    This summary is machine-generated.

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    Severe tetanus can cause peripheral neuropathy, affecting nerves like the ulnar and median. Electrophysiological studies confirm nerve damage, with recovery noted around ten weeks post-trismus onset.

    Area of Science:

    • Neurology
    • Infectious Diseases

    Background:

    • Tetanus is a serious bacterial infection with potential neurological complications.
    • Peripheral neuropathy is a recognized but less-studied manifestation of severe tetanus.

    Purpose of the Study:

    • To investigate the clinical and electrophysiological evidence of peripheral nerve involvement in severe tetanus.
    • To characterize the pattern and recovery of neuropathy in tetanus patients.

    Main Methods:

    • Studied 34 severe tetanus cases.
    • Clinical examination for neurological deficits.
    • Electrophysiological studies including nerve conduction velocity (NCV) and electromyography (EMG).

    Main Results:

    • Peripheral neuropathy observed in 27 of 34 patients, often asymmetrical.

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  • Commonly affected nerves included ulnar, median, and lateral popliteal.
  • Electrophysiology revealed denervation potentials, reduced motor/sensory conduction velocities, and decreased sensory amplitudes.
  • Nerve conduction showed improvement around 10 weeks after trismus onset.

    • Conclusions:

    • Severe tetanus is associated with significant peripheral nerve involvement.
    • Clinical and electrophysiological findings support a diagnosis of peripheral neuropathy in tetanus.
    • Nerve function typically recovers over several weeks to months.