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Toxic membrane fractions from Mycoplasma fermentans

Infection and Immunity
|December 1, 1971
PubMed

Insights

A Mycoplasma fermentans isolate caused lethal toxicity in mice. The toxic factor, located in reaggregated cell membranes, was a heat-stable lipid-protein complex, not lipids or proteins alone.

Area of Science:

  • Microbiology
  • Immunology
  • Toxicology

Background:

  • Mycoplasma fermentans is a bacterium known to cause various infections.
  • Certain strains of Mycoplasma fermentans can induce toxic effects in mammalian hosts.
  • Understanding the specific components responsible for Mycoplasma-induced toxicity is crucial for developing therapeutic strategies.

Purpose of the Study:

  • To identify the component of Mycoplasma fermentans responsible for lethal toxicity in mice.
  • To characterize the nature of the toxic factor present in the bacterium.

Main Methods:

  • Induction of toxicity in mice using viable and inactivated Mycoplasma fermentans cells, lysates, membranes, cytoplasm, and extracted lipids.
  • Detergent solubilization and Mg(2+)-mediated reaggregation of membrane components.
  • Fractionation of membrane components using acetone washes and ammonium sulfate precipitation.
  • Analysis of protein composition using polyacrylamide gel electrophoresis.
  • Assessment of the toxic factor's stability to heat, Pronase, trypsin, and lipase.

Main Results:

  • A specific isolate of Mycoplasma fermentans (strain K10) induced a lethal toxicity syndrome in mice.
  • Whole cell lysates and isolated membranes were toxic, while cytoplasm was not.
  • A toxic lipid-protein complex was formed when membranes were dissolved and reaggregated.
  • Isolated lipids or protein-rich fractions were non-toxic.
  • The toxic factor was heat-stable but degraded by Pronase, trypsin, and lipase, suggesting a protein component is involved.

Conclusions:

  • The lethal toxicity induced by this Mycoplasma fermentans isolate is mediated by a heat-stable lipid-protein complex found in the cell membranes.
  • Neither lipids nor proteins alone account for the observed toxicity.
  • The toxic factor's sensitivity to proteases and lipase indicates a crucial role for protein in its activity.

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