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Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function.

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Functionally distinct NMDA receptors mediate horizontal connectivity within layer 4 of mouse barrel cortex.

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Activation of protein kinase C increases neuronal excitability by regulating persistent Na+ current in mouse neocortical slices.

Journal of neurophysiology·1998

Kinetics of slow inactivation of persistent sodium current in layer V neurons of mouse neocortical slices.

Journal of neurophysiology·1996

Slow inactivation of Na+ current and slow cumulative spike adaptation in mouse and guinea-pig neocortical neurones in slices.

The Journal of physiology·1996

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Detection of intracerebral hemorrhage in putrefied pig brains using computed tomography, magnetic resonance imaging and a urine test strip.

Forensic science, medicine, and pathology·2026

Comparison of Expert Reads Versus Local Clinical Reads of Amyloid PET Scans in the IDEAS Study.

Journal of nuclear medicine : official publication, Society of Nuclear Medicine·2026

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Journal of medical virology·2026

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International journal of nanomedicine·2026

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Asian elephants (Elephas maximus) demonstrate inhibitory control in a detour task.

PloS one·2026

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Local anesthetic drug-induced seizures in rhesus monkeys

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Xenotransplantation of human intestine into mouse abdomen or subcutaneous tissue: Novel platforms for the study of the human enteric nervous system.

Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function.

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Activation of protein kinase C increases neuronal excitability by regulating persistent Na+ current in mouse neocortical slices.

Kinetics of slow inactivation of persistent sodium current in layer V neurons of mouse neocortical slices.

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