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The effects of maternal protein deprivation on renal development and function in neonatal rats

Insights

Maternal protein deprivation (PD) during nursing, not gestation, significantly impacts offspring kidney function and weight. This impaired renal transport of organic acids and bases, delaying maturation in developing kidneys.

Area of Science:

  • Nephrology
  • Developmental Biology
  • Nutritional Science

Background:

  • Maternal nutrition during gestation and lactation is crucial for fetal and neonatal development.
  • Protein deprivation (PD) can impact organ development and function.
  • Understanding the specific effects of PD on renal development is essential for public health.

Purpose of the Study:

  • To investigate the effects of pre- and/or postnatal maternal protein deprivation (PD) on the development of renal function in offspring.
  • To differentiate the impact of PD during gestation versus lactation on kidney development and function.

Main Methods:

  • Pregnant rats were fed control (24% protein) or low-protein (8-10%) diets during gestation and lactation.
  • Offspring were cross-fostered at birth to create four experimental groups: control-control, control-PD, PD-control, PD-PD.
  • Renal transport functions, organic acid and base transport, and alpha-aminoisobutyric acid accumulation were quantified in vitro.

Main Results:

  • Prenatal PD had negligible effects on body and kidney growth, while PD during nursing significantly impacted these parameters.
  • Renal transport capacity for organic acids and bases was depressed in pups nursed by PD dams.
  • Maturation of the renal organic acid transport system was delayed in pups exposed to PD during nursing.
  • Alpha-aminoisobutyric acid accumulation was enhanced in 10-day-old rats with both pre- and postnatal PD.

Conclusions:

  • Postnatal maternal protein deprivation during lactation has a more significant impact on renal functional development than prenatal exposure.
  • PD during nursing impairs renal transport of organic acids and bases and delays system maturation.
  • Renal gluconeogenic and ammoniagenic capacities were not affected by the dietary manipulations.

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