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[Autoimmunological receptor diseases].

A Fontana

    Schweizerische Medizinische Wochenschrift
    |March 10, 1979
    PubMed
    Summary
    This summary is machine-generated.

    Autoimmune diseases can disrupt cell receptor function. In conditions like Graves' disease, specific antibodies block hormone receptors, leading to illness.

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    Area of Science:

    • Molecular Biology
    • Immunology
    • Endocrinology

    Context:

    • Cellular communication relies on specific chemical signals binding to cell receptors.
    • Receptor function is crucial for initiating appropriate biological responses.
    • Autoimmune reactions targeting hormone receptors can impair normal physiological processes.

    Purpose:

    • To explore the mechanisms by which autoimmune reactions affect hormone receptor function.
    • To identify the role of immunoglobulins in disrupting receptor-mediated signaling.
    • To investigate the association of receptor-blocking immunoglobulins with specific autoimmune disorders.

    Summary:

    • Autoimmune responses can generate immunoglobulins that interfere with hormone binding to cell receptors.
    • These immunoglobulins can either abnormally stimulate or block receptor-mediated intracellular events.

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  • Such immunoglobulin activity has been observed in Graves' disease, myasthenia gravis, insulin resistance with acanthosis nigricans, and a seizure disorder linked to primary IgA deficiency.

    • Impact:

    • Highlights the pathogenic role of autoantibodies in endocrine and neurological diseases.
    • Provides insights into the molecular basis of receptor dysfunction in autoimmune conditions.
    • Suggests potential diagnostic or therapeutic targets related to receptor-binding immunoglobulins.