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Bipiperidyl mustard, a new obesifying agent in the mouse

Science (New York, N.Y.)
|August 26, 1966
PubMed

Insights

Bipiperidyl mustard induces rapid lipid deposition and obesity in mice via an alkylation mechanism. Its effects, similar to gold thioglucose, can be reversed by sulfhydryl compounds and involve hypothalamic lesions.

Area of Science:

  • Pharmacology
  • Neuroscience
  • Biochemistry

Background:

  • Bipiperidyl mustard is an alkylating agent.
  • Obesity is a complex metabolic disorder.
  • Gold thioglucose is known to induce obesity via hypothalamic lesions.

Purpose of the Study:

  • To investigate the effects of bipiperidyl mustard on lipid deposition and obesity in mice.
  • To explore the mechanism of action of bipiperidyl mustard.
  • To compare the effects of bipiperidyl mustard with gold thioglucose.

Main Methods:

  • Single-dose administration of bipiperidyl mustard to mice.
  • Observation of lipid deposition and body weight changes.
  • Assessment of responses to food restriction and refeeding.
  • Administration of sulfhydryl compounds as a counteraction.
  • Preliminary histological examination of hypothalamic lesions.

Main Results:

  • Bipiperidyl mustard caused rapid and extensive lipid deposition, leading to obesity.
  • The drug's effects were counteracted by sulfhydryl compounds.
  • Responses to dietary manipulations mimicked those seen with gold thioglucose.
  • Preliminary findings indicated ventromedial hypothalamic lesions in treated mice.

Conclusions:

  • Bipiperidyl mustard induces obesity through an alkylation reaction.
  • The mechanism of action shares similarities with gold thioglucose.
  • Hypothalamic ventromedial lesions are implicated in bipiperidyl mustard-induced obesity.

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