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Myelin and microsomes from rhesus monkey spinal cord: isolation and effects of trauma

Neurological Research
|January 1, 1980
PubMed

Insights

Rhesus monkey spinal cord myelin is more fragile than brain myelin, fragmenting into smaller vesicles during preparation. This fragility is evident in increased degraded myelin and altered myelin vesicle proportions after spinal cord trauma.

Area of Science:

  • Neuroscience
  • Biochemistry
  • Cell Biology

Background:

  • Myelin, the protective sheath around nerve fibers, is crucial for neural function.
  • Understanding myelin's structural integrity is vital, especially in the context of spinal cord injuries.
  • Differences in myelin composition and fragility between the brain and spinal cord are not fully understood.

Purpose of the Study:

  • To investigate and compare the fragility of myelin from rhesus monkey spinal cord and brain.
  • To analyze the biochemical and compositional differences between spinal cord and brain myelin.

Main Methods:

  • Differential centrifugation of rhesus monkey spinal cord and brain dispersions to obtain large (crude mitochondrial plus nuclear) and small (crude microsomal) particle fractions.
  • Isolation of myelin from these preparative fractions.
  • Analysis of myelin distribution, degradation products, lipid composition, protein content, and enzyme activity (2',3'-cyclic nucleotide phosphodiesterase).

Main Results:

  • Spinal cord myelin exhibited greater fragility, with nearly half found in the small particle fraction compared to brain myelin.
  • Larger amounts of partially degraded myelin were observed in spinal cord preparations.
  • Spinal cord myelin had lower protein content and reduced 2',3'-cyclic nucleotide phosphodiesterase specific activity than brain myelin.
  • Lipid compositions of spinal cord and brain microsomes showed some differences.

Conclusions:

  • Rhesus monkey spinal cord myelin is inherently more fragile than brain myelin.
  • Spinal cord myelin fragmentation leads to increased small myelin vesicles and degraded myelin upon dispersion.
  • Myelin fragility differences may have implications for understanding spinal cord injury pathology.

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