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[Tardive dyskinesias].

G Mignot

    L'Encephale
    |January 1, 1983
    PubMed
    Summary
    This summary is machine-generated.

    Tardive dyskinesia, a condition often developing during neuroleptic treatment, is linked to dopamine receptor changes. Research reviews its prevalence, characteristics, and treatment options based on this presumed cause.

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    Area of Science:

    • Neuroscience
    • Pharmacology
    • Movement Disorders

    Context:

    • Tardive dyskinesia (TD) is a common adverse effect of long-term neuroleptic medication.
    • Understanding TD's prevalence and risk factors is crucial for patient management.
    • The relationship between TD and other extrapyramidal symptoms requires further elucidation.

    Purpose:

    • To review the prevalence, risk factors, and clinical characteristics of tardive dyskinesia.
    • To explore the association between tardive dyskinesia and other extrapyramidal disorders.
    • To evaluate treatment strategies for tardive dyskinesia in light of its presumed pathophysiology.

    Summary:

    • Tardive dyskinesia (TD) frequently emerges during neuroleptic treatment, characterized by involuntary movements.

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  • The leading hypothesis suggests TD results from neuroleptic-induced dopamine receptor hypersensitivity.
  • This review examines TD's epidemiology, clinical presentation, and treatment efficacy based on the dopamine hypothesis.

    • Impact:

    • Provides a comprehensive overview of tardive dyskinesia for clinicians and researchers.
    • Highlights the importance of dopamine receptor sensitivity in TD pathophysiology.
    • Informs clinical practice regarding the management and treatment of neuroleptic-induced movement disorders.