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Renal function in long-term minoxidil-treated patients

Insights

Minoxidil treatment for refractory hypertension can lead to worsening kidney function in some patients, indicated by increased serum creatinine. Elevated norepinephrine levels may play a role in this renal disease progression.

Area of Science:

  • Nephrology
  • Cardiology
  • Pharmacology

Background:

  • Refractory hypertension poses significant challenges in patient management.
  • Minoxidil, in combination with sympathetic suppressants and diuretics, is used for severe hypertension.
  • The long-term effects of this regimen on renal function require further investigation.

Purpose of the Study:

  • To assess the impact of long-term minoxidil, sympathetic suppressants, and diuretics on renal function in hypertensive patients.
  • To investigate the relationship between neuroendocrine factors and renal disease progression in this patient cohort.

Main Methods:

  • Longitudinal monitoring of renal function (serum creatinine) in 20 patients over 2-7 years.
  • Measurement of plasma norepinephrine and plasma renin activity.
  • Correlation analysis between renal function, blood pressure control, therapy duration, and neuroendocrine markers.

Main Results:

  • Nine of 20 patients experienced a significant increase in serum creatinine (>1 mg/dl), with 4 progressing to hemodialysis.
  • Eleven patients showed no significant renal function decline; 3 with prior malignant hypertension and renal failure experienced reversal.
  • Higher plasma norepinephrine and lower plasma renin activity were observed in patients with renal disease progression.
  • Serum creatinine correlated with plasma norepinephrine levels.

Conclusions:

  • Long-term minoxidil-based therapy can be associated with progressive renal disease in a subset of refractory hypertensive patients.
  • Elevated plasma norepinephrine, potentially due to increased release rather than impaired renal clearance, may be linked to renal deterioration.
  • Further research is needed to elucidate the causal relationship between elevated norepinephrine and renal disease progression in hypertension.

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