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A cell membrane "gp70" associated with Fv-4 gene: immunological characterization, and tissue and strain distribution

Virology
|February 1, 1984
PubMed

Insights

Mice with the Fv-4r gene resist ecotropic murine leukemia virus (MuLV) infection. A unique gp70 molecule on Fv-4r mouse cells is linked to this resistance, suggesting a novel antiviral mechanism.

Area of Science:

  • Virology
  • Immunogenetics

Background:

  • The Fv-4r gene confers dominant resistance in mice against ecotropic murine leukemia virus (MuLV) infection.
  • Understanding the molecular basis of Fv-4r-mediated resistance is crucial for developing antiviral strategies.

Purpose of the Study:

  • To investigate the molecular nature of the Fv-4r resistance gene.
  • To identify the specific viral or cellular products associated with Fv-4r resistance.

Main Methods:

  • Analysis of cell surface gp70 expression in Fv-4r resistant mice.
  • Immunological characterization of Fv-4r-associated gp70 using specific antisera.
  • Detection of Fv-4r gp70 across various tissues and comparison of electrophoretic mobility.

Main Results:

  • A novel gp70 molecule, related to the MuLV env-gene product, was identified on cells from Fv-4r mice.
  • This Fv-4r gp70 exhibits unique antigenic determinants, distinct from other MuLV gp70s, and is recognized by Fv-4 specific antisera.
  • Fv-4r gp70 expression was detected in thymus, spleen, lymph nodes, bone marrow, and embryo fibroblasts, with tissue-specific electrophoretic mobility differences.

Conclusions:

  • The Fv-4r resistance allele is associated with the expression of a unique cell membrane gp70 molecule.
  • This Fv-4r-specific gp70 is a strong candidate for mediating resistance to ecotropic MuLV infection in mice.

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