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On disturbance of homeostasis in organ-cultured tissue

In Vitro
|August 1, 1984
PubMed

Insights

Rat mesentery tissue culture demonstrated spontaneous mast cell histamine release and increased fibroblast and mesothelial cell proliferation. These changes were influenced by culture medium serum concentration and may be linked.

Area of Science:

  • Tissue Engineering and Regenerative Medicine
  • Cell Biology
  • Histamine Signaling

Background:

  • Organ culture techniques are crucial for studying tissue homeostasis and cellular responses in vitro.
  • Mast cells play a significant role in inflammatory and homeostatic processes through histamine release.
  • Fibroblast and mesothelial cell proliferation are key indicators of tissue health and response to culture conditions.

Purpose of the Study:

  • To evaluate the homeostasis of rat mesentery tissue during organ culture.
  • To quantify spontaneous mast cell histamine release and fibroblast/mesothelial cell proliferation in cultured mesentery.
  • To investigate the impact of culture medium composition, specifically serum concentration, on these cellular events.

Main Methods:

  • Intact rat mesentery was cultured in Eagle's minimum essential medium with varying serum concentrations.
  • Spontaneous mast cell histamine release was measured over time using quantitative assays.
  • Fibroblast and mesothelial cell proliferation was assessed at 48 hours via cytofluorometric DNA quantitation.

Main Results:

  • Spontaneous histamine release from mast cells increased over time, reaching approximately 50% at 48 hours.
  • Tissue culture induced a significant increase in fibroblast and mesothelial cell proliferation, dependent on serum concentration.
  • Mast cell secretagogues (48/80, polymyxin B) induced rapid histamine release and augmented proliferation in serum-containing media.

Conclusions:

  • Rat mesentery organ culture leads to time-dependent mast cell histamine release and increased cell proliferation.
  • Serum concentration in the culture medium significantly influences both histamine release and cell proliferation.
  • Mast cell secretion may be causally linked to the observed spontaneous increase in cell proliferation, warranting further investigation.

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