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Bordetella pertussis respiratory tract infection in the mouse: pathophysiological responses

Insights

This study shows that intranasal infection with Bordetella pertussis in mice causes physiological changes like hypothermia and elevated leukocytes. These reactions, observed in a potential whooping cough model, vary with infection severity and mouse factors.

Area of Science:

  • Microbiology
  • Immunology
  • Pathophysiology

Background:

  • Whooping cough (pertussis) is a significant respiratory illness caused by Bordetella pertussis.
  • Understanding the host-pathogen interactions and disease progression is crucial for developing effective treatments and vaccines.
  • Mouse models are essential for studying infectious diseases, but a comprehensive model for pertussis pathophysiology is needed.

Purpose of the Study:

  • To investigate the pathophysiological effects of intranasal Bordetella pertussis infection in mice.
  • To determine the relationship between infection dose, lung infectivity, and observed physiological reactions.
  • To evaluate the suitability of the intranasally infected mouse as a model for studying whooping cough.

Main Methods:

  • Mice were infected intranasally with varying doses of live Bordetella pertussis.
  • Evaluated parameters included body weight, spleen size, body temperature, blood glucose, leukocyte count, and serum insulin levels.
  • Histamine sensitization was also assessed to measure disease severity.

Main Results:

  • Lethally infected mice exhibited weight loss, spleen atrophy, hypothermia, hypoglycemia, elevated leukocytes, and increased serum insulin.
  • Sublethally infected mice showed less severe symptoms, including spleen enlargement, normal temperature, and elevated leukocytes and insulin.
  • Reaction intensity correlated with lung infectivity, with hypothermia and leukocytosis showing a strong correlation. Serum insulin levels were linked to leukocytosis.

Conclusions:

  • The intranasally infected mouse model demonstrates dose-dependent pathophysiological changes relevant to whooping cough.
  • The observed reactions, including hypothermia, leukocytosis, and altered insulin levels, provide insights into pertussis pathogenesis.
  • This model shows promise for further research into whooping cough mechanisms and potential interventions.

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