Cellular senescence: cell proliferation and its control

Same author

A reproducible procedure for primary culture and subsequent maintenance of multiple lines of human skin fibroblasts.

Age·2013

Same author

Use of the fibroblast model in the study of cellular senescence.

Methods in molecular medicine·2012

Same author

SA beta Gal staining: biomarker or delusion.

Experimental gerontology·2005

Same author

Lack of Elk-1 phosphorylation and dysregulation of the extracellular regulated kinase signaling pathway in senescent human fibroblast.

Experimental cell research·2001

Same author

Loss of HuR is linked to reduced expression of proliferative genes during replicative senescence.

Molecular and cellular biology·2001

Same author

Is increased arachidonic acid release a cause or a consequence of replicative senescence?

Experimental gerontology·2001

Same journal

The biology of human aging.

Advances in pathobiology·1980

Same journal

Evolution of human longevity.

Advances in pathobiology·1980

Same journal

Approaches to drug selectivity in cancer therapy on the basis of differences between membranes of normal vs. neoplastic cells.

Advances in pathobiology·1980

Same journal

Long-term regulation of amino acid and hexose transport in cultured animal cells.

Advances in pathobiology·1980

Same journal

Genetic analysis of the human cell-surface antigens expressed by a human-Chinese hamster somatic cell hybrid.

Advances in pathobiology·1980

Same journal

Glycolipid changes associated with oncogenesis and ontogenesis, and the inhibition of the process of transformation by monovalent anti-glycolipid antibodies.

Advances in pathobiology·1980

Related Experiment Videos

Cellular senescence: cell proliferation and its control

Related Concept Videos

A reproducible procedure for primary culture and subsequent maintenance of multiple lines of human skin fibroblasts.

Use of the fibroblast model in the study of cellular senescence.

SA beta Gal staining: biomarker or delusion.

Lack of Elk-1 phosphorylation and dysregulation of the extracellular regulated kinase signaling pathway in senescent human fibroblast.

Loss of HuR is linked to reduced expression of proliferative genes during replicative senescence.

Is increased arachidonic acid release a cause or a consequence of replicative senescence?

The biology of human aging.

Evolution of human longevity.

Approaches to drug selectivity in cancer therapy on the basis of differences between membranes of normal vs. neoplastic cells.

Long-term regulation of amino acid and hexose transport in cultured animal cells.

Genetic analysis of the human cell-surface antigens expressed by a human-Chinese hamster somatic cell hybrid.

Glycolipid changes associated with oncogenesis and ontogenesis, and the inhibition of the process of transformation by monovalent anti-glycolipid antibodies.

Related Experiment Videos

Cellular senescence: cell proliferation and its control

Related Concept Videos

Related Articles

A reproducible procedure for primary culture and subsequent maintenance of multiple lines of human skin fibroblasts.

Use of the fibroblast model in the study of cellular senescence.

SA beta Gal staining: biomarker or delusion.

Lack of Elk-1 phosphorylation and dysregulation of the extracellular regulated kinase signaling pathway in senescent human fibroblast.

Loss of HuR is linked to reduced expression of proliferative genes during replicative senescence.

Is increased arachidonic acid release a cause or a consequence of replicative senescence?

The biology of human aging.

Evolution of human longevity.

Approaches to drug selectivity in cancer therapy on the basis of differences between membranes of normal vs. neoplastic cells.

Long-term regulation of amino acid and hexose transport in cultured animal cells.

Genetic analysis of the human cell-surface antigens expressed by a human-Chinese hamster somatic cell hybrid.

Glycolipid changes associated with oncogenesis and ontogenesis, and the inhibition of the process of transformation by monovalent anti-glycolipid antibodies.