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An additional role for the outer nuclear membrane in the morphogenesis of herpes simplex virus

Intervirology
|January 1, 1980
PubMed

Insights

Herpes simplex virus may fuse with the outer nuclear membrane in MRC-5 cells, releasing naked capsids into the cytoplasm. This suggests a novel role for the nuclear membrane in viral replication before cell lysis.

Area of Science:

  • Virology
  • Cell Biology
  • Microbiology

Background:

  • Herpes simplex virus (HSV) infection involves complex interactions with host cell machinery.
  • The precise mechanisms of HSV capsid transport and envelopment within host cells are not fully understood.
  • The role of the nuclear envelope in viral egress and replication requires further elucidation.

Purpose of the Study:

  • To investigate the potential involvement of the outer nuclear membrane in herpes simplex virus replication.
  • To identify the ultrastructural basis for the early appearance of naked cytoplasmic capsids during HSV infection.
  • To explore a novel pathway for viral particle release and cytoplasmic entry.

Main Methods:

  • Ultrastructural analysis of MRC-5 cells infected with herpes simplex virus.
  • Time-course observation of viral particle formation and localization postinfection (p.i.).
  • Microscopy techniques to visualize the nuclear envelope and viral structures.

Main Results:

  • Observed physical continuity between the outer nuclear membrane and viral envelopes in the perinuclear cisterna.
  • Evidence suggesting fusion between the nuclear membrane and viral envelopes, leading to capsid envelopment.
  • Naked cytoplasmic capsids appeared as early as 6 hours postinfection (p.i.).
  • Nuclear membrane disruption was observed significantly later, at 24 hours p.i.

Conclusions:

  • The outer nuclear membrane may directly participate in the production of naked cytoplasmic capsids through a fusion event.
  • This fusion mechanism provides an alternative pathway for viral particles to enter the cytoplasm, preceding nuclear membrane breakdown.
  • Suggests a previously unrecognized role for the outer nuclear membrane in herpes simplex virus life cycle and replication.

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