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Transposition of Tn 1 to the Rhizobium meliloti genome

Molecular & General Genetics : MGG
|January 1, 1980
PubMed

Insights

Researchers used a modified plasmid to introduce the ampicillin-resistant transposon Tn1 into Rhizobium meliloti. This revealed specific insertion sites in the genome and symbiotic plasmids, without causing auxotrophy.

Area of Science:

  • Microbiology
  • Genetics
  • Molecular Biology

Background:

  • The IncP1 plasmid RP4 derivative, carrying Mucts62, was constructed in Escherichia coli.
  • This hybrid plasmid, RP4::Mucts62, proved unstable in Rhizobium meliloti.

Purpose of the Study:

  • To utilize the unstable hybrid plasmid as a vehicle for transposon delivery.
  • To investigate the insertion specificity of the ampicillin-resistance transposon Tn1 in the R. meliloti genome.

Main Methods:

  • Transposon mutagenesis using Tn1 delivered via the RP4::Mucts62 plasmid.
  • Cotransductional analysis with phage DF2 to identify chromosomal insertion hot spots.
  • Plasmid-curing experiments via heat treatment to assess Tn1 insertion in symbiotic plasmids.

Main Results:

  • Transposition of Tn1 into R. meliloti did not result in auxotrophic mutants, indicating specific insertion.
  • Two chromosomal hot spots for Tn1 insertion were identified.
  • Symbiotic plasmids of R. meliloti were found to contain at least one site for Tn1 insertion.

Conclusions:

  • The RP4::Mucts62 plasmid serves as an effective tool for Tn1 delivery into R. meliloti.
  • Tn1 insertion into the R. meliloti genome exhibits site specificity, with identified chromosomal and symbiotic plasmid targets.

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