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Reversible inhibition of cytomegalovirus replication by phosphonoformate

Intervirology
|January 1, 1980
PubMed

Insights

Phosphonoformate (PFA) inhibits Cytomegalovirus (CMV) replication by affecting antigen production. However, the CMV genome persists and reactivates after PFA removal, indicating non-abortive infection.

Area of Science:

  • Virology
  • Immunology

Background:

  • Cytomegalovirus (CMV) is a significant human pathogen.
  • Antiviral agents like phosphonoformate (PFA) are used to manage CMV infections.

Purpose of the Study:

  • To investigate the effect of PFA on CMV antigen expression and replication kinetics.
  • To determine if CMV infection is reversible after PFA treatment.

Main Methods:

  • Anticomplement immunofluorescence assays were used to detect CMV immediate early antigens (IEA) and early nuclear antigens (EA).
  • Microimmunofluorometry quantified antigen levels in individual infected nuclei.
  • Kinetics of antigen appearance and persistence were analyzed with and without PFA.

Main Results:

  • PFA specifically inhibited CMV-induced late antigens.
  • CMV antigens appeared in two waves (6 and 24 hours post-infection), a pattern maintained in the presence of PFA.
  • CMV-infected cells persisted for over 5 weeks with PFA, and viral replication resumed upon PFA removal.

Conclusions:

  • The CMV genome persists in infected cells during PFA treatment.
  • CMV infection is not abortive under PFA treatment and can reactivate.
  • PFA's antiviral action is reversible, highlighting the dynamic nature of CMV persistence.

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