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Polymyxin interaction with negatively charged lipid bilayer membranes and the competitive effect of Ca2+

Insights

Polymyxin-B binding to lipid membranes causes phase separation and alters lipid transitions. Calcium ions compete with polymyxin-B binding, with effectiveness varying by pH and lipid type.

Area of Science:

  • Biochemistry
  • Membrane Biophysics
  • Pharmacology

Background:

  • Polymyxin-B is a cationic antibiotic that interacts with bacterial membranes.
  • Understanding its binding to anionic phospholipids is crucial for its mechanism of action.

Purpose of the Study:

  • To investigate the binding of polymyxin-B to phosphatidic acid and phosphatidylglycerol membranes.
  • To explore the competitive binding of calcium ions (Ca2+) with polymyxin-B.

Main Methods:

  • Fluorescence polarization studies were employed to analyze binding.
  • Competition experiments with Ca2+ were conducted at varying pH levels.

Main Results:

  • Polymyxin-B binding induced phase separation in mixed phosphatidic acid/phosphatidylcholine membranes, forming polymyxin-bound phosphatidic acid domains.
  • Ca2+ effectively displaced polymyxin-B from mixed membranes at pH 9.0 but was ineffective at pH 5.0.
  • Polymyxin-B binding to phosphatidylglycerol membranes lowered the lipid phase transition temperature by 10°C, with a noncooperative binding stoichiometry of 1:5 (polymyxin:phosphatidylglycerol).
  • Ca2+ reduced phosphatidylglycerol binding to polymyxin-B by approximately 20% without complete displacement.

Conclusions:

  • Polymyxin-B binding to anionic phospholipids is concentration-dependent and can induce membrane domain formation.
  • Calcium ions act as competitors for polymyxin-B binding, with pH-dependent efficacy.
  • Polymyxin-B incorporation significantly affects the biophysical properties of phosphatidylglycerol-containing membranes.

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