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Mycoplasma-induced BALB/c 3T3 collagenase is a mammalian enzyme

Insights

Mycoplasma orale infection of mouse cells induces them to produce a vertebrate-type collagenase. This enzyme, distinct from bacterial collagenases, degrades specific collagen types, indicating a host-cell response to infection.

Area of Science:

  • Biochemistry
  • Cell Biology
  • Microbiology

Background:

  • BALB/c 3T3 cells infected with Mycoplasma orale accumulate a collagenase.
  • The origin of this collagenase (host vs. microbe) was previously unclear.

Purpose of the Study:

  • To partially purify and characterize the collagenase.
  • To determine if the collagenase is of mammalian or bacterial origin.

Main Methods:

  • Partial purification of the enzyme.
  • Characterization of enzyme properties: activation, inhibitor sensitivity, molecular mass, substrate specificity, and reaction products.
  • Comparison with known vertebrate and bacterial collagenases.

Main Results:

  • The enzyme exhibited properties similar to vertebrate collagenases regarding purification, activation, inhibitor sensitivity, and molecular mass.
  • Substrate specificity and reaction products were typical of vertebrate collagenases, distinguishing it from bacterial collagenases.
  • The enzyme preferentially degraded type III and type I collagen, with limited activity against type II and type IV collagen.

Conclusions:

  • The collagenase produced by mycoplasma-infected BALB/c 3T3 cells is a mammalian-type enzyme.
  • The enzyme is a product of the mouse genome, not the mycoplasma.
  • Mycoplasma orale infection induces BALB/c 3T3 mouse fibroblasts to produce and secrete collagenase.

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